Can intracellular signalling pathways predict developmental abnormalities? - Wikidata - wikimedia - TriplyDB

Can intracellular signalling pathways predict developmental abnormalities?

Can intracellular signalling pathways predict developmental abnormalities?

http://www.wikidata.org/entity/Q42442037

about

Antenatal glucocorticoids and programming of the developing CNS.

P2860

Q33858306-7E8727A4-D5C8-43CE-87A5-CFD1BCCA33E2

P2860

Can intracellular signalling pathways predict developmental abnormalities?

http://www.wikidata.org/entity/Q42442037

description

1996 nî lūn-bûn

@nan

1996年の論文

@ja

1996年論文

@yue

1996年論文

@zh-hant

1996年論文

@zh-hk

1996年論文

@zh-mo

1996年論文

@zh-tw

1996年论文

@wuu

1996年论文

@zh

1996年论文

@zh-cn

name

Can intracellular signalling pathways predict developmental abnormalities?

@en

Can intracellular signalling pathways predict developmental abnormalities?

@nl

type

label

Can intracellular signalling pathways predict developmental abnormalities?

@en

Can intracellular signalling pathways predict developmental abnormalities?

@nl

prefLabel

Can intracellular signalling pathways predict developmental abnormalities?

@en

Can intracellular signalling pathways predict developmental abnormalities?

@nl

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Can intracellular signalling pathways predict developmental abnormalities?

@en

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C Lau

http://www.w3.org/2001/XMLSchema#string

F J Seidler

http://www.w3.org/2001/XMLSchema#string

S E Lappi

http://www.w3.org/2001/XMLSchema#string

T A Slotkin

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P2860

Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease

The c-Fos protein interacts with c-Jun/AP-1 to stimulate transcription of AP-1 responsive genes

c-fos and ornithine decarboxylase gene expression in brain as early markers of neurotoxicity

Effects of prenatal terbutaline exposure on cellular development in lung and liver of neonatal rat: ornithine decarboxylase activity and macromolecules

Corticosterone differentially regulates the expression of Gs alpha and Gi alpha messenger RNA and protein in rat cerebral cortex

Selective loss of noradrenergic phenotypic characters in neuroblasts of the rat embryo.

The role of cellular oncogenes in myogenesis and muscle cell hypertrophy.

Ontogenetic appearance and disappearance of tyrosine hydroxylase and catecholamines in the rat embryo

Dexamethasone attenuates kainate-induced AP-1 activation in rat brain.

Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro.

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115-122

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scholarly article

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10.3109/13547509609088679

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2

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1

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23888922

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