Common genetic variation in a basal promoter element alters DDAH2 expression in endothelial cells.
about
A functional variant of the dimethylarginine dimethylaminohydrolase-2 gene is associated with insulin sensitivitySequence variation in DDAH1 and DDAH2 genes is strongly and additively associated with serum ADMA concentrations in individuals with type 2 diabetes.Regulation of the ADMA-DDAH system in endothelial cells: a novel mechanism for the sterol response element binding proteins, SREBP1c and -2.Pilot study of the association of the DDAH2 -449G polymorphism with asymmetric dimethylarginine and hemodynamic shock in pediatric sepsis.Septic shock is correlated with asymmetrical dimethyl arginine levels, which may be influenced by a polymorphism in the dimethylarginine dimethylaminohydrolase II gene: a prospective observational study.Asymmetric dimethylarginine (ADMA): the silent transition from an 'uraemic toxin' to a global cardiovascular risk molecule.Genetic regulation of endothelial functionDDAH gene and cardiovascular risk.Arginine, arginine analogs and nitric oxide production in chronic kidney disease.Nitric oxide deficiency in chronic kidney disease.Asymmetric dimethylarginine inhibits HSP90 activity in pulmonary arterial endothelial cells: role of mitochondrial dysfunctionEndogenous nitric oxide synthase inhibitors in the biology of disease: markers, mediators, and regulators?Association of DDAH2 gene polymorphism with cardiovascular disease in Egyptian patients.Assessment of serum levels of asymmetric dimethylarginine, symmetric dimethylarginine and L-arginine in coronary artery disease.Ammonia Reduces Intracellular Asymmetric Dimethylarginine in Cultured Astrocytes Stimulating Its y⁺LAT2 Carrier-Mediated Loss.Hyper-activation of pp60Src limits nitric oxide signaling by increasing asymmetric dimethylarginine levels during acute lung injury.Association of the -449GC and -1151AC polymorphisms in the DDAH2 gene with asymmetric dimethylarginine and erythropoietin resistance in Chinese patients on maintenance hemodialysis.
P2860
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P2860
Common genetic variation in a basal promoter element alters DDAH2 expression in endothelial cells.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Common genetic variation in a ...... pression in endothelial cells.
@en
Common genetic variation in a ...... pression in endothelial cells.
@nl
type
label
Common genetic variation in a ...... pression in endothelial cells.
@en
Common genetic variation in a ...... pression in endothelial cells.
@nl
prefLabel
Common genetic variation in a ...... pression in endothelial cells.
@en
Common genetic variation in a ...... pression in endothelial cells.
@nl
P2093
P1476
Common genetic variation in a ...... xpression in endothelial cells
@en
P2093
C T L Tran
J M Leiper
P Vallance
P304
P356
10.1016/J.BBRC.2003.09.097
P577
2003-10-01T00:00:00Z