Characterization of the hipA7 allele of Escherichia coli and evidence that high persistence is governed by (p)ppGpp synthesis. - Wikidata - wikimedia - TriplyDB

Characterization of the hipA7 allele of Escherichia coli and evidence that high persistence is governed by (p)ppGpp synthesis.

Characterization of the hipA7 allele of Escherichia coli and evidence that high persistence is governed by (p)ppGpp synthesis.

http://www.wikidata.org/entity/Q42452904

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Comprehensive functional analysis of Mycobacterium tuberculosis toxin-antitoxin systems: implications for pathogenesis, stress responses, and evolution The RelA/SpoT homolog (RSH) superfamily: distribution and functional evolution of ppGpp synthetases and hydrolases across the tree of life Bacterial toxin-antitoxin systems: more than selfish entities?Bacterial persistence: a model of survival in changing environments Hypothetical functions of toxin-antitoxin systems Type II toxin: antitoxin systems. More than small selfish entities?Toxin-Antitoxin systems: their role in persistence, biofilm formation, and pathogenicity Culture history and population heterogeneity as determinants of bacterial adaptation: the adaptomics of a single environmental transition To be or not to be: regulation of restriction-modification systems and other toxin-antitoxin systems Direct binding targets of the stringent response alarmone (p)ppGpp Nongenetic individuality in the host-phage interaction Structure of the Escherichia coli Antitoxin MqsA (YgiT/b3021) Bound to Its Gene Promoter Reveals Extensive Domain Rearrangements and the Specificity of Transcriptional Regulation Role of Unusual P Loop Ejection and Autophosphorylation in HipA-Mediated Persistence and Multidrug Tolerance The HicA toxin from Burkholderia pseudomallei has a role in persister cell formation The bacterial antitoxin HipB establishes a ternary complex with operator DNA and phosphorylated toxin HipA to regulate bacterial persistence HipBA-promoter structures reveal the basis of heritable multidrug tolerance Structure, Biology, and Therapeutic Application of Toxin-Antitoxin Systems in Pathogenic Bacteria Microbial cell individuality and the underlying sources of heterogeneity Slow protein fluctuations explain the emergence of growth phenotypes and persistence in clonal bacterial populations Three Mycobacterium tuberculosis Rel toxin-antitoxin modules inhibit mycobacterial growth and are expressed in infected human macrophages PhoY2 but not PhoY1 is the PhoU homologue involved in persisters in Mycobacterium tuberculosis Wake me when it's over - Bacterial toxin-antitoxin proteins and induced dormancy Prokaryotic toxin-antitoxin stress response loci Biofilm-related infections: bridging the gap between clinical management and fundamental aspects of recalcitrance toward antibiotics.Single-cell protein induction dynamics reveals a period of vulnerability to antibiotics in persister bacteria.The chromosomal toxin gene yafQ is a determinant of multidrug tolerance for Escherichia coli growing in a biofilm.Regulation of phenotypic variability by a threshold-based mechanism underlies bacterial persistence.Effects of the twin-arginine translocase on the structure and antimicrobial susceptibility of Escherichia coli biofilms.GlpD and PlsB participate in persister cell formation in Escherichia coli.Why is long-term therapy required to cure tuberculosis?Role of global regulators and nucleotide metabolism in antibiotic tolerance in Escherichia coli.Ciprofloxacin causes persister formation by inducing the TisB toxin in Escherichia coli.The three RelE homologs of Mycobacterium tuberculosis have individual, drug-specific effects on bacterial antibiotic tolerance VapC toxins from Mycobacterium tuberculosis are ribonucleases that differentially inhibit growth and are neutralized by cognate VapB antitoxins.Plasmid addiction systems: perspectives and applications in biotechnology.Regulation of the Escherichia coli HipBA toxin-antitoxin system by proteolysis Large mutational target size for rapid emergence of bacterial persistence Control of Biofilms with the Fatty Acid Signaling Molecule cis-2-Decenoic Acid.What Is the Link between Stringent Response, Endoribonuclease Encoding Type II Toxin-Antitoxin Systems and Persistence?The fatty acid signaling molecule cis-2-decenoic acid increases metabolic activity and reverts persister cells to an antimicrobial-susceptible state.

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P2860

Characterization of the hipA7 allele of Escherichia coli and evidence that high persistence is governed by (p)ppGpp synthesis.

http://www.wikidata.org/entity/Q42452904

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2003 nî lūn-bûn

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Characterization of the hipA7 ...... overned by (p)ppGpp synthesis.

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Characterization of the hipA7 ...... igh persistence is governed by

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Characterization of the hipA7 ...... overned by (p)ppGpp synthesis.

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Characterization of the hipA7 ...... igh persistence is governed by

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Characterization of the hipA7 ...... overned by (p)ppGpp synthesis.

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Characterization of the hipA7 ...... igh persistence is governed by

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J.1365-2958.2003.03779.X

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Molecular Microbiology

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Characterization of the hipA7 ...... overned by (p)ppGpp synthesis.

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Shaleen B Korch

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Thomas A Henderson

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Thomas M Hill

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P2860

Tight regulation, modulation, and high-level expression by vectors containing the arabinose PBAD promoter

sfi-independent filamentation in Escherichia coli Is lexA dependent and requires DNA damage for induction.

New method for generating deletions and gene replacements in Escherichia coli

Conditional impairment of cell division and altered lethality in hipA mutants of Escherichia coli K-12.

A locus affecting nucleoid segregation in Salmonella typhimurium.

Molecular cloning and expression of hipA, a gene of Escherichia coli K-12 that affects frequency of persistence after inhibition of murein synthesis.

Site-specific deletions of chromosomally located DNA segments with the multimer resolution system of broad-host-range plasmid RP4

Direct correlation between overproduction of guanosine 3',5'-bispyrophosphate (ppGpp) and penicillin tolerance in Escherichia coli.

Autoregulation of hip, an operon that affects lethality due to inhibition of peptidoglycan or DNA synthesis

Structure and organization of hip, an operon that affects lethality due to inhibition of peptidoglycan or DNA synthesis.

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