Endothelin-1-induced arrhythmogenic Ca2+ signaling is abolished in atrial myocytes of inositol-1,4,5-trisphosphate(IP3)-receptor type 2-deficient mice.
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Increased InsP3Rs in the junctional sarcoplasmic reticulum augment Ca2+ transients and arrhythmias associated with cardiac hypertrophyWide long lasting perinuclear Ca2+ release events generated by an interaction between ryanodine and IP3 receptors in canine Purkinje cellsIP3 receptor-mediated Ca2+ release in naive CD4 T cells dictates their cytokine programAstrocyte and Neuronal Plasticity in the Somatosensory SystemCalcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approachesIon Channels in the HeartOptogenetic and Chemogenetic Approaches for Studying Astrocytes and GliotransmittersThe MLCK-mediated alpha1-adrenergic inotropic effect in atrial myocardium is negatively modulated by PKCepsilon signalingProperties of Ca2+ sparks revealed by four-dimensional confocal imaging of cardiac muscleAstrocyte IP3R2-dependent Ca(2+) signaling is not a major modulator of neuronal pathways governing behaviorAmerican Heart Association atrial fibrillation research summit: a conference report from the American Heart AssociationTwo-photon microscopy as a tool to study blood flow and neurovascular coupling in the rodent brain.Ryanodine receptors are uncoupled from contraction in rat vena cavaAstrocytic calcium release mediates peri-infarct depolarizations in a rodent stroke model.Inositol-1,4,5-trisphosphate-mediated spontaneous activity in mouse embryonic stem cell-derived cardiomyocytes.CaMKII regulation of cardiac ryanodine receptors and inositol triphosphate receptors.Calcium release from intra-axonal endoplasmic reticulum leads to axon degeneration through mitochondrial dysfunctionNo contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure overload hypertrophy.Extracellular matrix remodeling in atrial fibrosis: mechanisms and implications in atrial fibrillation.Simultaneous loss of phospholipase Cδ1 and phospholipase Cδ3 causes cardiomyocyte apoptosis and cardiomyopathy.Conditions and constraints for astrocyte calcium signaling in the hippocampal mossy fiber pathway.Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure.Inositol trisphosphate receptor Ca2+ release channels.Hippocampal short- and long-term plasticity are not modulated by astrocyte Ca2+ signaling.The IP3 receptor regulates cardiac hypertrophy in response to select stimuli.Astrocytes mediate in vivo cholinergic-induced synaptic plasticitySpecific disruption of astrocytic Ca2+ signaling pathway in vivo by adeno-associated viral transduction.Astrocytic Gq-GPCR-linked IP3R-dependent Ca2+ signaling does not mediate neurovascular coupling in mouse visual cortex in vivo.Activation of NFATc1 is directly mediated by IP3 in adult cardiac myocytesAstrocyte inositol triphosphate receptor type 2 and cytosolic phospholipase A2 alpha regulate arteriole responses in mouse neocortical brain slices.IP3 receptor type 2 deficiency is associated with a secretory defect in the pancreatic acinar cell and an accumulation of zymogen granules.Regulation of multidrug resistance-associated protein 2 by calcium signaling in mouse liver.Inositol 1,4,5-trisphosphate receptor type II (InsP3R-II) is reduced in obese mice, but metabolic homeostasis is preserved in mice lacking InsP3R-II.Calpain-dependent cleavage of junctophilin-2 and T-tubule remodeling in a mouse model of reversible heart failureCalcium and arrhythmogenesis.Endoplasmic reticulum Ca(2+) handling in excitable cells in health and diseaseRecent advances in the molecular pathophysiology of atrial fibrillation.The involvement of TRPC3 channels in sinoatrial arrhythmiasEffects of thrombin and thrombin receptor activation on cardiac function after acute myocardial infarctionThe type 2 inositol 1,4,5-trisphosphate receptor, emerging functions for an intriguing Ca²⁺-release channel.
P2860
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P2860
Endothelin-1-induced arrhythmogenic Ca2+ signaling is abolished in atrial myocytes of inositol-1,4,5-trisphosphate(IP3)-receptor type 2-deficient mice.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@en
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@nl
type
label
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@en
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@nl
prefLabel
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@en
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@nl
P2093
P1433
P1476
Endothelin-1-induced arrhythmo ...... eceptor type 2-deficient mice.
@en
P2093
Aleksey V Zima
Farah Sheikh
Lothar A Blatter
Xiaodong Li
P304
P356
10.1161/01.RES.0000172556.05576.4C
P577
2005-06-02T00:00:00Z