The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells. - Wikidata - wikimedia - TriplyDB

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

http://www.wikidata.org/entity/Q42482430

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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination miRNEST database: an integrative approach in microRNA search and annotation Pancreatic cancer stem cell markers and exosomes - the incentive push The Role of microRNAs in the Diagnosis and Treatment of Pancreatic Adenocarcinoma MicroRNA Regulation of Human Breast Cancer Stem Cells Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis The Roles of Epithelial-to-Mesenchymal Transition (EMT) and Mesenchymal-to-Epithelial Transition (MET) in Breast Cancer Bone Metastasis: Potential Targets for Prevention and Treatment Interactions between E-cadherin and microRNA deregulation in head and neck cancers: the potential interplay MiR-200, a new star miRNA in human cancer From fly wings to targeted cancer therapies: a centennial for notch signaling Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition The microRNA-200 family: small molecules with novel roles in cancer development, progression and therapy The Regulatory Role of MicroRNAs in EMT and Cancer Activated K-ras and INK4a/Arf deficiency cooperate during the development of pancreatic cancer by activation of Notch and NF-κB signaling pathways MicroRNA-147 induces a mesenchymal-to-epithelial transition (MET) and reverses EGFR inhibitor resistance Comprehensive logic based analyses of Toll-like receptor 4 signal transduction pathway EMT and dissemination precede pancreatic tumor formation The role of miRNAs in bone metastasis and their significance in the detection of bone metastasis: a review of the published data.PLA2G7 associates with hormone receptor negativity in clinical breast cancer samples and regulates epithelial-mesenchymal transition in cultured breast cancer cells The mir-675-5p regulates the progression and development of pancreatic cancer via the UBQLN1-ZEB1-mir200 axis ZEB1 regulates glioma stemness through LIF repression.Epithelial-to-mesenchymal transition and the cancer stem cell phenotype: insights from cancer biology with therapeutic implications for colorectal cancer.Endometrial miR-200c is altered during transformation into cancerous states and targets the expression of ZEBs, VEGFA, FLT1, IKKβ, KLF9, and FBLN5.Histone deacetylases, microRNA and leptin crosstalk in pancreatic cancer.Clinicopathological and prognostic implications of the miR-200 family in patients with epithelial ovarian cancer.ZEB1 induces ER-α promoter hypermethylation and confers antiestrogen resistance in breast cancer.The Role of Fibroblasts in Pancreatic Cancer: Extracellular Matrix Versus Paracrine Factors.Members of the microRNA-200 family are promising therapeutic targets in cancer.Sweating the small stuff: microRNAs and genetic changes define pancreatic cancer.Epithelial/mesenchymal plasticity: how have quantitative mathematical models helped improve our understanding?Intestinal epithelial barrier disruption through altered mucosal microRNA expression in human immunodeficiency virus and simian immunodeficiency virus infections Role of non-coding RNAs in pancreatic cancer: the bane of the microworld.Concise review: breast cancer stem cells: regulatory networks, stem cell niches, and disease relevance microRNA-141 regulates BMI1 expression and induces senescence in human diploid fibroblasts.Cancer stem cells: involvement in pancreatic cancer pathogenesis and perspectives on cancer therapeutics.MicroRNA alterations and associated aberrant DNA methylation patterns across multiple sample types in oral squamous cell carcinoma.Downregulation of endothelial microRNA-200b supports cutaneous wound angiogenesis by desilencing GATA binding protein 2 and vascular endothelial growth factor receptor 2 A pituitary homeobox 2 (Pitx2):microRNA-200a-3p:β-catenin pathway converts mesenchymal cells to amelogenin-expressing dental epithelial cells The epigenetics of epithelial-mesenchymal plasticity in cancer.DCLK1 regulates pluripotency and angiogenic factors via microRNA-dependent mechanisms in pancreatic cancer.

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The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

http://www.wikidata.org/entity/Q42482430

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name

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

@en

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

@nl

type

label

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

@en

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

@nl

prefLabel

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

@en

The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells.

@nl

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The EMBO Journal

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The ZEB1/miR-200 feedback loop controls Notch signalling in cancer cells

@en

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Arno Dimmler

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Elke Firat

http://www.w3.org/2001/XMLSchema#string

Gabriele Niedermann

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Gerhard Faller

http://www.w3.org/2001/XMLSchema#string

Jörg Schubert

http://www.w3.org/2001/XMLSchema#string

Karolina Bajdak

http://www.w3.org/2001/XMLSchema#string

Simone Brabletz

http://www.w3.org/2001/XMLSchema#string

Simone Meidhof

http://www.w3.org/2001/XMLSchema#string

Thomas Brabletz

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Ulrike Burk

http://www.w3.org/2001/XMLSchema#string

P2860

Jagged1-mediated Notch activation induces epithelial-to-mesenchymal transition through Slug-induced repression of E-cadherin.

Core signaling pathways in human pancreatic cancers revealed by global genomic analyses

The miR-200 family inhibits epithelial-mesenchymal transition and cancer cell migration by direct targeting of E-cadherin transcriptional repressors ZEB1 and ZEB2

The ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

The epithelial-mesenchymal transition generates cells with properties of stem cells

Let-7 and miR-200 microRNAs: guardians against pluripotency and cancer progression

Epithelial-mesenchymal transitions in development and disease

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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770-782

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10.1038/EMBOJ.2010.349

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4

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30

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2011-01-11T00:00:00Z

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49745678

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21224848

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3041948

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