N-cadherin is overexpressed in Crohn's stricture fibroblasts and promotes intestinal fibroblast migration.
about
Mechanisms that mediate the development of fibrosis in patients with Crohn's disease.Anti-fibrogenic effect of PPAR-γ agonists in human intestinal myofibroblasts.A comprehensive analysis of adiponectin QTLs using SNP association, SNP cis-effects on peripheral blood gene expression and gene expression correlation identified novel metabolic syndrome (MetS) genes with potential role in carcinogenesis and systemSignificant contribution of TRPC6 channel-mediated Ca2+ influx to the pathogenesis of Crohn's disease fibrotic stenosis.Intestinal myofibroblast TRPC6 channel may contribute to stenotic fibrosis in Crohn's disease.
P2860
N-cadherin is overexpressed in Crohn's stricture fibroblasts and promotes intestinal fibroblast migration.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@en
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@nl
type
label
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@en
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@nl
prefLabel
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@en
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@nl
P2093
P2860
P356
P1476
N-cadherin is overexpressed in ...... testinal fibroblast migration.
@en
P2093
Catherine Sweeney
John P Burke
Michael F Cunningham
P Ronan O'Connell
P2860
P304
P356
10.1002/IBD.21543
P577
2011-02-01T00:00:00Z