Elevation of resting mitochondrial membrane potential of neural cells by cyclosporin A, BAPTA-AM, and bcl-2.
about
Visualizing cell death in experimental focal cerebral ischemia: promises, problems, and perspectives.An interaction between Bcl-xL and the voltage-dependent anion channel (VDAC) promotes mitochondrial Ca2+ uptake.Enhanced mesenchymal stem cell survival induced by GATA-4 overexpression is partially mediated by regulation of the miR-15 familyNADPH oxidase 4 deficiency increases tubular cell death during acute ischemic reperfusion injury.Bax translocation to mitochondria subsequent to a rapid loss of mitochondrial membrane potential.Effect of ATP and Bax on the apoptosis of Eimeria tenella host cells.
P2860
Elevation of resting mitochondrial membrane potential of neural cells by cyclosporin A, BAPTA-AM, and bcl-2.
description
2000 nî lūn-bûn
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name
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@en
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@nl
type
label
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@en
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@nl
prefLabel
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@en
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@nl
P2093
P2860
P1476
Elevation of resting mitochond ...... sporin A, BAPTA-AM, and bcl-2.
@en
P2093
Kowaltowski AJ
Russell JT
P2860
P304
P356
10.1152/AJPCELL.2000.279.3.C852
P577
2000-09-01T00:00:00Z