UCP1 and defense against oxidative stress. 4-Hydroxy-2-nonenal effects on brown fat mitochondria are uncoupling protein 1-independent.
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Loss of UCP2 attenuates mitochondrial dysfunction without altering ROS production and uncoupling activityMechanism of fatty-acid-dependent UCP1 uncoupling in brown fat mitochondriaUltraendurance exercise increases the production of reactive oxygen species in isolated mitochondria from human skeletal muscle.Role of the transmembrane potential in the membrane proton leak.Cold exposure promotes atherosclerotic plaque growth and instability via UCP1-dependent lipolysis.Fatty acids are key in 4-hydroxy-2-nonenal-mediated activation of uncoupling proteins 1 and 2Uncoupling protein 1 binds one nucleotide per monomer and is stabilized by tightly bound cardiolipin.Test systems to study the structure and function of uncoupling protein 1: a critical overview.High membrane potential promotes alkenal-induced mitochondrial uncoupling and influences adenine nucleotide translocase conformationMetabolically inert perfluorinated fatty acids directly activate uncoupling protein 1 in brown-fat mitochondriaStimulation of mitochondrial proton conductance by hydroxynonenal requires a high membrane potential.Detoxification reactions: relevance to aging.Improved health-span and lifespan in mtDNA mutator mice treated with the mitochondrially targeted antioxidant SkQ1.Mitochondrial ('mild') uncoupling and ROS production: physiologically relevant or not?The molecular features of uncoupling protein 1 support a conventional mitochondrial carrier-like mechanismUCP1 deficiency causes brown fat respiratory chain depletion and sensitizes mitochondria to calcium overload-induced dysfunctionUncoupling protein 1 decreases superoxide production in brown adipose tissue mitochondria.Mitochondrial reactive oxygen species and adipose tissue thermogenesis: Bridging physiology and mechanisms.Stress turns on the heat: Regulation of mitochondrial biogenesis and UCP1 by ROS in adipocytes.Mitochondrial uncoupling proteins: subtle regulators of cellular redox signaling.UCP1 inhibition in Cidea-overexpressing mice is physiologically counteracted by brown adipose tissue hyperrecruitment.Brown adipose tissue redox status in response to dietary-induced obesity-associated oxidative stress in male and female rats.
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P2860
UCP1 and defense against oxidative stress. 4-Hydroxy-2-nonenal effects on brown fat mitochondria are uncoupling protein 1-independent.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
UCP1 and defense against oxida ...... oupling protein 1-independent.
@en
UCP1 and defense against oxida ...... oupling protein 1-independent.
@nl
type
label
UCP1 and defense against oxida ...... oupling protein 1-independent.
@en
UCP1 and defense against oxida ...... oupling protein 1-independent.
@nl
prefLabel
UCP1 and defense against oxida ...... oupling protein 1-independent.
@en
UCP1 and defense against oxida ...... oupling protein 1-independent.
@nl
P2093
P2860
P50
P356
P1476
UCP1 and defense against oxida ...... coupling protein 1-independent
@en
P2093
Joris Hoeks
Natasa Petrovic
Tatiana V Kramarova
P2860
P304
13882-13893
P356
10.1074/JBC.M601387200
P407
P577
2006-03-16T00:00:00Z