Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
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No evidence for new dopaminergic neurons in the adult mammalian substantia nigraStriatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.A single intramuscular injection of rAAV-mediated mutant erythropoietin protects against MPTP-induced parkinsonism.Physical activity and environmental enrichment regulate the generation of neural precursors in the adult mouse substantia nigra in a dopamine-dependent manner.Quantitative assessment of gait and neurochemical correlation in a classical murine model of Parkinson's disease.Sequential Loss of LC Noradrenergic and Dopaminergic Neurons Results in a Correlation of Dopaminergic Neuronal Number to Striatal Dopamine Concentration.Increases in cytoplasmic dopamine compromise the normal resistance of the nucleus accumbens to methamphetamine neurotoxicityA phenotypic model recapitulating the neuropathology of Parkinson's disease.Neuronal NOS and cyclooxygenase-2 contribute to DNA damage in a mouse model of Parkinson disease.Activation of tyrosine hydroxylase mRNA translation by cAMP in midbrain dopaminergic neurons.N-acetylcysteine prevents loss of dopaminergic neurons in the EAAC1-/- mouseMicrodialysis in the rat striatum: effects of 24 h dexamethasone retrodialysis on evoked dopamine release and penetration injury.Neuroprotective Surgical Strategies in Parkinson's Disease: Role of Preclinical Data.The mouse MPTP model: gene expression changes in dopaminergic neurons.Lack of long-term changes in circadian, locomotor, and cognitive functions in acute and chronic MPTP (1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine) mouse models of Parkinson's disease.Metalloproteinase-9 contributes to inflammatory glia activation and nigro-striatal pathway degeneration in both mouse and monkey models of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced Parkinsonism.Exercise-Induced Neuroprotection of the Nigrostriatal Dopamine System in Parkinson's Disease.Targeted deletion of GD3 synthase protects against MPTP-induced neurodegeneration.Rhes Counteracts Dopamine Neuron Degeneration and Neuroinflammation Depending on Gender and Age.Conditioning Against the Pathology of Parkinson's disease
P2860
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P2860
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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2000年学术文章
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2000年学术文章
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name
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@en
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@nl
type
label
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@en
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@nl
prefLabel
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@en
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@nl
P2093
P1433
P1476
Spontaneous long-term compensatory dopaminergic sprouting in MPTP-treated mice.
@en
P2093
P2860
P304
P356
10.1002/1098-2396(20001201)38:3<363::AID-SYN16>3.0.CO;2-A
P50
P577
2000-12-01T00:00:00Z