Chloride channel ClC-2 modulates tight junction barrier function via intracellular trafficking of occludin.
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Molecular aspects of tight junction barrier functionClC-2 regulation of intestinal barrier function: Translation of basic science to therapeutic targetMegalencephalic leukoencephalopathy with subcortical cysts protein-1 modulates endosomal pH and protein trafficking in astrocytes: relevance to MLC disease pathogenesisLubiprostone improves intestinal permeability in humans, a novel therapy for the leaky gut: A prospective randomized pilot study in healthy volunteers.Lactobacillus casei reduces susceptibility to type 2 diabetes via microbiota-mediated body chloride ion influx.Contrasting effects of linaclotide and lubiprostone on restitution of epithelial cell barrier properties and cellular homeostasis after exposure to cell stressors.A comparison of linaclotide and lubiprostone dosing regimens on ion transport responses in human colonic mucosaMatrix metalloproteinase 9-induced increase in intestinal epithelial tight junction permeability contributes to the severity of experimental DSS colitisRole of autophagy in the regulation of epithelial cell junctionsChloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.Chloride channelopathies of ClC-2.Large Animal Models: The Key to Translational Discovery in Digestive Disease Research.Continuous endocytic recycling of tight junction proteins: how and why?The use of nonsteroidal anti-inflammatory drugs in critically ill horses.Regulatory-auxiliary subunits of CLC chloride channel-transport proteins.Chloride channel ClC- 2 enhances intestinal epithelial tight junction barrier function via regulation of caveolin-1 and caveolar trafficking of occludin.Myosin light chain kinase mediates intestinal barrier dysfunction via occludin endocytosis during anoxia/reoxygenation injury.Differentiation between human ClC-2 and CFTR Cl- channels with pharmacological agents.Indomethacin induces increase in gastric epithelial tight junction permeability via redistribution of occludin and activation of p38 MAPK in MKN-28 Cells.Mouse models of human ocular disease for translational research.Pharmaceutical Activation or Genetic Absence of ClC-2 Alters Tight Junctions During Experimental Colitis.Real-time monitoring of trans-epithelial electrical resistance in cultured intestinal epithelial cells: the barrier protection of water-soluble dietary fiber.Par-3 modulates intestinal epithelial barrier function through regulating intracellular trafficking of occludin and myosin light chain phosphorylation.
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P2860
Chloride channel ClC-2 modulates tight junction barrier function via intracellular trafficking of occludin.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
@zh-hant
name
Chloride channel ClC-2 modulat ...... lular trafficking of occludin.
@en
Chloride channel ClC-2 modulat ...... lular trafficking of occludin.
@nl
type
label
Chloride channel ClC-2 modulat ...... lular trafficking of occludin.
@en
Chloride channel ClC-2 modulat ...... lular trafficking of occludin.
@nl
prefLabel
Chloride channel ClC-2 modulat ...... lular trafficking of occludin.
@en
Chloride channel ClC-2 modulat ...... lular trafficking of occludin.
@nl
P2860
P1476
Chloride channel ClC-2 modulat ...... llular trafficking of occludin
@en
P2093
Prashant K Nighot
P2860
P304
P356
10.1152/AJPCELL.00072.2011
P577
2011-09-28T00:00:00Z