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Capture Hi-C identifies a novel causal gene, IL20RA, in the pan-autoimmune genetic susceptibility region 6q23Effect of the oral application of a highly selective MMP-13 inhibitor in three different animal models of rheumatoid arthritis.Association of circulating miR-223 and miR-16 with disease activity in patients with early rheumatoid arthritis.The mesenchymal stem cell marker CD248 (endosialin) is a negative regulator of bone formation in mice.Altered expression of microRNA-203 in rheumatoid arthritis synovial fibroblasts and its role in fibroblast activation.Synovial activation in rheumatoid arthritis.Expression and Regulation of PIWIL-Proteins and PIWI-Interacting RNAs in Rheumatoid Arthritis.Gene analysis for exploring the effects of drugs in rheumatoid arthritis.Proteinases in the joint: clinical relevance of proteinases in joint destructionTechnology insight: gene transfer and the design of novel treatments for rheumatoid arthritis.Is there a future for small molecule drugs in the treatment of rheumatic diseases?Educational needs and preferences of young European clinicians and physician researchers working in the field of rheumatology.Prospective new biological therapies for rheumatoid arthritis.Epigenetics in rheumatoid arthritis.TLRs and chronic inflammation.AAA-ATPase p97 suppresses apoptotic and autophagy-associated cell death in rheumatoid arthritis synovial fibroblasts.Inflammatory cytokines epigenetically regulate rheumatoid arthritis fibroblast-like synoviocyte activation by suppressing HDAC5 expressionWhat can we learn from epigenetics in the year 2009?Epigenetically-driven anatomical diversity of synovial fibroblasts guides joint-specific fibroblast functions.Inflammatory memories: is epigenetics the missing link to persistent stromal cell activation in rheumatoid arthritis?Epigenetic changes: the missing link.Nucleic acid-stimulated antigen-presenting cells trigger T cells to induce disease in a rat transfer model of inflammatory arthritis.The epigenetic architecture at gene promoters determines cell type-specific LPS tolerance.Genomic Responses of Mouse Synovial Fibroblasts During Tumor Necrosis Factor-Driven Arthritogenesis Greatly Mimic Those in Human Rheumatoid Arthritis.Epigenetic biomarkers in rheumatology - the future?Epigenetics in the pathogenesis of RA.Why location matters - site-specific factors in rheumatic diseases.Identification of microRNA-221/222 and microRNA-323-3p association with rheumatoid arthritis via predictions using the human tumour necrosis factor transgenic mouse model.The Natural Polyphenol Epigallocatechin Gallate Protects Intervertebral Disc Cells from Oxidative Stress.Social media use among young rheumatologists and basic scientists: results of an international survey by the Emerging EULAR Network (EMEUNET).Safety concerns on the development of novel therapeutic drugs.SIRT6 regulates the cigarette smoke-induced signalling in rheumatoid arthritis synovial fibroblasts.The pattern-recognition receptor nucleotide-binding oligomerization domain--containing protein 1 promotes production of inflammatory mediators in rheumatoid arthritis synovial fibroblasts.Down-regulation of microRNA-34a* in rheumatoid arthritis synovial fibroblasts promotes apoptosis resistance.Pre-B cell colony-enhancing factor/visfatin, a new marker of inflammation in rheumatoid arthritis with proinflammatory and matrix-degrading activities.Synovial fibroblasts in 2017.Inhibition of fibroblast activation protein and dipeptidylpeptidase 4 increases cartilage invasion by rheumatoid arthritis synovial fibroblasts.Functional autoantibodies against serpin E2 in rheumatoid arthritis.Identification of a genetic variant for joint damage progression in autoantibody-positive rheumatoid arthritis.Time course of coagulation parameters, cytokines and adhesion molecules in Plasmodium falciparum malaria.
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hulumtuese
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հետազոտող
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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Caroline Ospelt
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P21
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0000 0003 9619 6679
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P31
P496
0000-0002-9151-4650
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