Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
about
Platelet-derived growth factor: a key factor in the pathogenesis of graves' ophthalmopathy and potential target for treatmentCurrent concepts in the molecular pathogenesis of thyroid-associated ophthalmopathy.Graves' ophthalmopathyThe Role of Thyrotropin Receptor Activation in Adipogenesis and Modulation of Fat Phenotype.A stimulatory thyrotropin receptor antibody (M22) and thyrotropin increase interleukin-6 expression and secretion in Graves' orbital preadipocyte fibroblasts.A stimulatory TSH receptor antibody enhances adipogenesis via phosphoinositide 3-kinase activation in orbital preadipocytes from patients with Graves' ophthalmopathy.Thyrotropin and obesity: increased adipose triglyceride content through glycerol-3-phosphate acyltransferase 3.Mutations that silence constitutive signaling activity in the allosteric ligand-binding site of the thyrotropin receptor.Human longevity is characterised by high thyroid stimulating hormone secretion without altered energy metabolismRole of extrathyroidal TSHR expression in adipocyte differentiation and its association with obesityImmunopathogenesis of Graves' ophthalmopathy: the role of the TSH receptor.The thyroid, the eyes and the gut: a possible connection.Unique attributes of orbital fibroblasts and global alterations in IGF-1 receptor signaling could explain thyroid-associated ophthalmopathy.Role of insulin-like growth factor-1 (IGF-1) pathway in the pathogenesis of Graves' orbitopathy.Recent insights into the pathogenesis and management of thyroid-associated ophthalmopathyRegulation of Lymphocyte Function by PPARgamma: Relevance to Thyroid Eye Disease-Related InflammationTSH stimulates adipogenesis in mouse embryonic stem cellsGsalpha signalling suppresses PPARgamma2 generation and inhibits 3T3L1 adipogenesis.Effects of prostaglandin F(2α) on adipocyte biology relevant to graves' orbitopathy.A modified ELISA accurately measures secretion of high molecular weight hyaluronan (HA) by Graves' disease orbital cells.Thyrotropin receptor activation increases hyaluronan production in preadipocyte fibroblasts: contributory role in hyaluronan accumulation in thyroid dysfunction.Inhibiting thyrotropin/insulin-like growth factor 1 receptor crosstalk to treat Graves' ophthalmopathy: studies in orbital fibroblasts in vitro.Fat pad-derived mesenchymal stem cells as a potential source for cell-based adipose tissue repair strategies.Modulation of expression of somatostatin receptor subtypes in Graves' ophthalmopathy orbits: relevance to novel analogs.TSH/TSHR Signaling Suppresses Fatty Acid Synthase (FASN) Expression in Adipocytes.
P2860
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P2860
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@en
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@nl
type
label
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@en
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@nl
prefLabel
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@en
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@nl
P2093
P2860
P356
P1476
Biological effects of thyrotropin receptor activation on human orbital preadipocytes.
@en
P2093
Carol A Paddon
Dagmar Fuhrer
Dominika Janus
Glynn Baker
Marian Ludgate
P2860
P304
P356
10.1167/IOVS.06-0596
P407
P577
2006-12-01T00:00:00Z