TRAF-1, -2, -3, -5, and -6 are induced in atherosclerotic plaques and differentially mediate proinflammatory functions of CD40L in endothelial cells.
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Roles of tumor necrosis factor receptor associated factor 3 (TRAF3) and TRAF5 in immune cell functionsThe role of costimulatory receptors of the tumour necrosis factor receptor family in atherosclerosisMAP-Kinase Activated Protein Kinase 2 Links Endothelial Activation and Monocyte/macrophage Recruitment in ArteriogenesisDeficient CD40-TRAF6 signaling in leukocytes prevents atherosclerosis by skewing the immune response toward an antiinflammatory profileCD40L and Its Receptors in Atherothrombosis-An UpdateCannabinoid receptor 2 signaling does not modulate atherogenesis in miceTumor necrosis factor receptor associated factor 6 is not required for atherogenesis in mice and does not associate with atherosclerosis in humans.CD40 is essential in the upregulation of TRAF proteins and NF-kappaB-dependent proinflammatory gene expression after arterial injuryHuman native lipoprotein-induced de novo DNA methylation is associated with repression of inflammatory genes in THP-1 macrophagescIAP1-dependent TRAF2 degradation regulates the differentiation of monocytes into macrophages and their response to CD40 ligandCD40L deficiency attenuates diet-induced adipose tissue inflammation by impairing immune cell accumulation and production of pathogenic IgG-antibodies.Immortalized liver endothelial cells: a cell culture model for studies of motility and angiogenesis.Tumor necrosis factor receptor-associated factor 1 (TRAF1) deficiency attenuates atherosclerosis in mice by impairing monocyte recruitment to the vessel wallCrucial role of CD40 signaling in vascular wall cells in neointimal formation and vascular remodeling after vascular interventionsTRAF6 inhibits proangiogenic signals in endothelial cells and regulates the expression of vascular endothelial growth factor.TRAF6 protein couples Toll-like receptor 4 signaling to Src family kinase activation and opening of paracellular pathway in human lung microvascular endothelia.Genetically driven target tissue overexpression of CD40: a novel mechanism in autoimmune disease.TRAF1 is a key mediator for hepatic ischemia/reperfusion injurySystematic analysis of the molecular mechanism underlying atherosclerosis using a text mining approachTRAF1 is a critical regulator of cerebral ischaemia-reperfusion injury and neuronal death.Peripheral venous congestion causes inflammation, neurohormonal, and endothelial cell activation.The CD40-CD40L system in cardiovascular disease.Helicobacter pylori inhibits the cleavage of TRAF1 via a CagA-dependent mechanism.The Upregulation of TRAF1 Induced by Helicobacter pylori Plays an Antiapoptotic Effect on the Infected Cells.Blockade of CD40-TRAF2,3 or CD40-TRAF6 is sufficient to inhibit pro-inflammatory responses in non-haematopoietic cells.Tumor necrosis factor receptor-associated factor-6 and ribosomal S6 kinase intracellular pathways link the angiotensin II AT1 receptor to the phosphorylation and activation of the IkappaB kinase complex in vascular smooth muscle cells.Soluble CD40 ligand profiles in patients with septic shock.TRAF3IP2 mediates atherosclerotic plaque development and vulnerability in ApoE(-/-) mice.TRAF5 and TRAF3IP2 gene polymorphisms are associated with Behçet's disease and Vogt-Koyanagi-Harada syndrome: a case-control study.Endothelial cell toll-like receptor 4 regulates fibrosis-associated angiogenesis in the liver.Directing TRAF-ic: cell-specific TRAF6 signaling in chronic inflammation and atherosclerosis.Intrapancreatic injection of human bone marrow-derived mesenchymal stem/stromal cells alleviates hyperglycemia and modulates the macrophage state in streptozotocin-induced type 1 diabetic mice.The CD40-TRAF6 axis is the key regulator of the CD40/CD40L system in neointima formation and arterial remodeling.Tumor necrosis factor receptor-associated factor 5 is an essential mediator of ischemic brain infarction.TRAF molecules in inflammation and inflammatory diseases.
P2860
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P2860
TRAF-1, -2, -3, -5, and -6 are induced in atherosclerotic plaques and differentially mediate proinflammatory functions of CD40L in endothelial cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells.
@en
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells.
@nl
type
label
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells.
@en
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells.
@nl
prefLabel
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells.
@en
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells.
@nl
P2093
P50
P1476
TRAF-1, -2, -3, -5, and -6 are ...... of CD40L in endothelial cells
@en
P2093
Erdyni Tsitsikov
Joanna Jagielska
Lindsey MacFarlane
Masanori Aikawa
Peter Libby
Sandra Ernst
Udo Bavendiek
Uwe Schönbeck
P304
P356
10.1161/ATVBAHA.107.140566
P407
P577
2007-03-01T00:00:00Z