Upstream stimulatory factors binding to an E box motif in the R region of the bovine leukemia virus long terminal repeat stimulates viral gene expression.
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Mechanisms of leukemogenesis induced by bovine leukemia virus: prospects for novel anti-retroviral therapies in humanHuman prolyl-4-hydroxylase alpha(I) transcription is mediated by upstream stimulatory factorsComparison of bovine leukemia virus (BLV) and CMV promoter-driven reporter gene expression in BLV-infected and non-infected cellsTranscription factor binding sites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivityThe AP-1 binding sites located in the pol gene intragenic regulatory region of HIV-1 are important for viral replicationChromatin disruption in the promoter of bovine leukemia virus during transcriptional activationPotentiation of tumor necrosis factor-induced NF-kappa B activation by deacetylase inhibitors is associated with a delayed cytoplasmic reappearance of I kappa B alpha.Defibrinated bovine plasma inhibits retroviral transcription by blocking p52 activation of the NFkappaB element in the long terminal repeatCharacterization of new RNA polymerase III and RNA polymerase II transcriptional promoters in the Bovine Leukemia Virus genome.Repression of Human T-lymphotropic virus type 1 Long Terminal Repeat sense transcription by Sp1 recruitment to novel Sp1 binding sites.Overlapping CRE and E box motifs in the enhancer sequences of the bovine leukemia virus 5' long terminal repeat are critical for basal and acetylation-dependent transcriptional activity of the viral promoter: implications for viral latency.Genome-wide identification, classification, and functional analysis of the basic helix-loop-helix transcription factors in the cattle, Bos Taurus.DNA cytosine methylation in the bovine leukemia virus promoter is associated with latency in a lymphoma-derived B-cell line: potential involvement of direct inhibition of cAMP-responsive element (CRE)-binding protein/CRE modulator/activation transcrDeacetylase inhibitors and the viral transactivator TaxBLV synergistically activate bovine leukemia virus gene expression via a cAMP-responsive element- and cAMP-responsive element-binding protein-dependent mechanism.Characterization of novel Bovine Leukemia Virus (BLV) antisense transcripts by deep sequencing reveals constitutive expression in tumors and transcriptional interaction with viral microRNAs.Transcriptional regulation of the bovine leukemia virus promoter by the cyclic AMP-response element modulator tau isoform.
P2860
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P2860
Upstream stimulatory factors binding to an E box motif in the R region of the bovine leukemia virus long terminal repeat stimulates viral gene expression.
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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2001年学术文章
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2001年学术文章
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name
Upstream stimulatory factors b ...... mulates viral gene expression.
@en
Upstream stimulatory factors b ...... mulates viral gene expression.
@nl
type
label
Upstream stimulatory factors b ...... mulates viral gene expression.
@en
Upstream stimulatory factors b ...... mulates viral gene expression.
@nl
prefLabel
Upstream stimulatory factors b ...... mulates viral gene expression.
@en
Upstream stimulatory factors b ...... mulates viral gene expression.
@nl
P2093
P2860
P50
P356
P1476
Upstream stimulatory factors b ...... mulates viral gene expression.
@en
P2093
Arsene Burny
Claire Calomme
Louis Droogmans
Thi Lien-Anh Nguyen
P2860
P304
P356
10.1074/JBC.M107441200
P407
P577
2001-12-10T00:00:00Z