Adenylyl cyclase activity and function are decreased in rat cardiac fibroblasts after myocardial infarction.
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Uridine triphosphate (UTP) induces profibrotic responses in cardiac fibroblasts by activation of P2Y2 receptorsReduced collagen deposition in infarcted myocardium facilitates induced pluripotent stem cell engraftment and angiomyogenesis for improvement of left ventricular function.Improved function of the failing rat heart by regulated expression of insulin-like growth factor I via intramuscular gene transfer.Increase in cellular cyclic AMP concentrations reverses the profibrogenic phenotype of cardiac myofibroblasts: a novel therapeutic approach for cardiac fibrosis.Unanticipated signaling events associated with cardiac adenylyl cyclase gene transfer.Adenylyl cyclase regulation in heart failure due to myocardial infarction in rats.
P2860
Adenylyl cyclase activity and function are decreased in rat cardiac fibroblasts after myocardial infarction.
description
2007 nî lūn-bûn
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2007年の論文
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2007年学术文章
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name
Adenylyl cyclase activity and ...... s after myocardial infarction.
@en
Adenylyl cyclase activity and ...... s after myocardial infarction.
@nl
type
label
Adenylyl cyclase activity and ...... s after myocardial infarction.
@en
Adenylyl cyclase activity and ...... s after myocardial infarction.
@nl
prefLabel
Adenylyl cyclase activity and ...... s after myocardial infarction.
@en
Adenylyl cyclase activity and ...... s after myocardial infarction.
@nl
P2093
P2860
P1476
Adenylyl cyclase activity and ...... s after myocardial infarction.
@en
P2093
David M Roth
Hemal H Patel
James S Swaney
Matthew Spellman
N Chin Lai
Paul A Insel
Utako Yokoyama
P2860
P304
P356
10.1152/AJPHEART.00739.2007
P577
2007-09-14T00:00:00Z