Effects of myeloid differentiation primary response gene 88 (MyD88) activation on Helicobacter infection in vivo and induction of a Th17 response.
about
IL-22 negatively regulates Helicobacter pylori-induced CCL20 expression in gastric epithelial cellsGenetic polymorphisms and tissue expression of interleukin-22 associated with risk and therapeutic response of gastric mucosa-associated lymphoid tissue lymphomaIn vivo treatment of Helicobacter pylori infection with liposomal linolenic acid reduces colonization and ameliorates inflammationEffect of myeloid differentiation primary response gene 88 on expression profiles of genes during the development and progression of Helicobacter-induced gastric cancerGli1 deletion prevents Helicobacter-induced gastric metaplasia and expansion of myeloid cell subsetsDeficiency of the myeloid differentiation primary response molecule MyD88 leads to an early and rapid development of Helicobacter-induced gastric malignancy.Inflammation, immunity, and vaccines for Helicobacter pylori infection.Helicobacter spp. other than H. pylori.Helicobacter pylori induces increased expression of the vitamin d receptor in immune responses.
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Effects of myeloid differentiation primary response gene 88 (MyD88) activation on Helicobacter infection in vivo and induction of a Th17 response.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年學術文章
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2011年學術文章
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2011年學術文章
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name
Effects of myeloid differentia ...... induction of a Th17 response.
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Effects of myeloid differentiation primary response gene 88
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type
label
Effects of myeloid differentia ...... induction of a Th17 response.
@en
Effects of myeloid differentiation primary response gene 88
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prefLabel
Effects of myeloid differentia ...... induction of a Th17 response.
@en
Effects of myeloid differentiation primary response gene 88
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Effects of myeloid differentia ...... induction of a Th17 response.
@en
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Barry Rickman
Donald G Guiney
Marygorret Obonyo
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P304
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10.1111/J.1523-5378.2011.00861.X
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2011-10-01T00:00:00Z