Pancreatic cancers rely on a novel glutamine metabolism pathway to maintain redox balance.
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KRAS Mutant Pancreatic Cancer: No Lone Path to an Effective TreatmentDysregulated metabolism contributes to oncogenesisNovel therapeutic targets of tumor metabolismROS homeostasis and metabolism: a dangerous liason in cancer cellsAnalysis and interpretation of transcriptomic data obtained from extended Warburg effect genes in patients with clear cell renal cell carcinoma.The complex landscape of pancreatic cancer metabolism[Ferroptosis, a new form of cell death relevant to the medical treatment of cancer].Cancerous epithelial cell lines shed extracellular vesicles with a bimodal size distribution that is sensitive to glutamine inhibitionMetabolic phenotypes in pancreatic cancer.GOT1/AST1 expression status as a prognostic biomarker in pancreatic ductal adenocarcinoma.Targeting glutamine metabolism sensitizes pancreatic cancer to PARP-driven metabolic catastrophe induced by ß-lapachoneMetabolic Reprogramming of Pancreatic Cancer Mediated by CDK4/6 Inhibition Elicits Unique VulnerabilitiesEnvironment Impacts the Metabolic Dependencies of Ras-Driven Non-Small Cell Lung CancerThe glucose transporter GLUT1 is required for ErbB2-induced mammary tumorigenesis.Metabolic Dependencies in RAS-Driven Cancers.The greedy nature of mutant RAS: a boon for drug discovery targeting cancer metabolism?Mutant KRAS associated malic enzyme 1 expression is a predictive marker for radiation therapy response in non-small cell lung cancer.Metabolic Interactions in the Tumor Microenvironment.Glutamine-dependent α-ketoglutarate production regulates the balance between T helper 1 cell and regulatory T cell generation.Glutaminase is essential for the growth of triple-negative breast cancer cells with a deregulated glutamine metabolism pathway and its suppression synergizes with mTOR inhibition.HIF-2α regulates non-canonical glutamine metabolism via activation of PI3K/mTORC2 pathway in human pancreatic ductal adenocarcinoma.Macropinocytosis: A Metabolic Adaptation to Nutrient Stress in Cancer.Matrine suppresses KRAS-driven pancreatic cancer growth by inhibiting autophagy-mediated energy metabolism.LAT2 regulates glutamine-dependent mTOR activation to promote glycolysis and chemoresistance in pancreatic cancer
P2860
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P2860
Pancreatic cancers rely on a novel glutamine metabolism pathway to maintain redox balance.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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name
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@en
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@nl
type
label
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@en
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@nl
prefLabel
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@en
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@nl
P2860
P356
P1433
P1476
Pancreatic cancers rely on a n ...... way to maintain redox balance.
@en
P2093
Alec C Kimmelman
Jaekyoung Son
P2860
P304
P356
10.4161/CC.25307
P577
2013-06-10T00:00:00Z