Cholesterol efflux potential and antiinflammatory properties of high-density lipoprotein after treatment with niacin or anacetrapib.
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Antagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosisNiacin Alternatives for Dyslipidemia: Fool's Gold or Gold Mine? Part II: Novel Niacin MimeticsStructural Insights into High Density Lipoprotein: Old Models and New FactsNiacin, an old drug with a new twistRegression of atherosclerosis: insights from animal and clinical studiesNew Era of Lipid-Lowering DrugsWill Lipidation of ApoA1 through Interaction with ABCA1 at the Intestinal Level Affect the Protective Functions of HDL?Niacin Therapy, HDL Cholesterol, and Cardiovascular Disease: Is the HDL Hypothesis Defunct?Cholesterol, inflammation and innate immunityGPR109A and vascular inflammationThe therapeutic role of niacin in dyslipidemia managementAnacetrapib, a Novel CETP Inhibitor: Pursuing a New Approach to Cardiovascular Risk ReductionPrimary prevention of coronary heart disease: integration of new data, evolving views, revised goals, and role of rosuvastatin in management. A comprehensive survey.Cholesterol efflux and atheroprotection: advancing the concept of reverse cholesterol transport.Triglyceride-rich lipoproteins and high-density lipoprotein cholesterol in patients at high risk of cardiovascular disease: evidence and guidance for managementCholesteryl ester transfer protein inhibition as a strategy to reduce cardiovascular risk.miR33 inhibition overcomes deleterious effects of diabetes mellitus on atherosclerosis plaque regression in mice.Xanthohumol prevents atherosclerosis by reducing arterial cholesterol content via CETP and apolipoprotein E in CETP-transgenic mice.High-density lipoprotein (HDL) particle subpopulations in heterozygous cholesteryl ester transfer protein (CETP) deficiency: maintenance of antioxidative activity.Effect of apoA-I Mutations in the Capacity of Reconstituted HDL to Promote ABCG1-Mediated Cholesterol Efflux.Anacetrapib reduces progression of atherosclerosis, mainly by reducing non-HDL-cholesterol, improves lesion stability and adds to the beneficial effects of atorvastatin.Increased atherosclerosis in mice with vascular ATP-binding cassette transporter G1 deficiency--brief report.eNOS activation by HDL is impaired in genetic CETP deficiency.Anacetrapib promotes reverse cholesterol transport and bulk cholesterol excretion in Syrian golden hamsters.Paraoxonase-3 is depleted from the high-density lipoproteins of autoimmune disease patients with subclinical atherosclerosis.Changes in lipoprotein subfraction concentration and composition in healthy individuals treated with the CETP inhibitor anacetrapib.Novel HDL-directed pharmacotherapeutic strategies.Atherosclerosis: current status of prevention and treatment.Using bioinformatics and systems genetics to dissect HDL-cholesterol genetics in an MRL/MpJ x SM/J intercrossEffect of Extended-Release Niacin on High-Density Lipoprotein (HDL) Functionality, Lipoprotein Metabolism, and Mediators of Vascular Inflammation in Statin-Treated Patients.Genetic variants in TLR2 and TLR4 are associated with markers of monocyte activation: the Atherosclerosis Risk in Communities MRI StudyTargeting high density lipoproteins in the prevention of cardiovascular disease?Carboxyl-Terminal Cleavage of Apolipoprotein A-I by Human Mast Cell Chymase Impairs Its Anti-Inflammatory Properties.Niacin Therapy Increases High-Density Lipoprotein Particles and Total Cholesterol Efflux Capacity But Not ABCA1-Specific Cholesterol Efflux in Statin-Treated Subjects.Cholesteryl Ester Transfer Protein Inhibition With Anacetrapib Decreases Fractional Clearance Rates of High-Density Lipoprotein Apolipoprotein A-I and Plasma Cholesteryl Ester Transfer Protein.Future of cholesteryl ester transfer protein (CETP) inhibitors: a pharmacological perspective.Defining the Safety of Anacetrapib, a CETP Inhibitor, in Patients at High Risk for Coronary Heart Disease: the DEFINE study.In vivo effects of anacetrapib on preβ HDL: improvement in HDL remodeling without effects on cholesterol absorption.Lecithin cholesterol acyltransferase: an anti- or pro-atherogenic factor?Molecular sources of residual cardiovascular risk, clinical signals, and innovative solutions: relationship with subclinical disease, undertreatment, and poor adherence: implications of new evidence upon optimizing cardiovascular patient outcomes.
P2860
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P2860
Cholesterol efflux potential and antiinflammatory properties of high-density lipoprotein after treatment with niacin or anacetrapib.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@en
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@nl
type
label
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@en
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@nl
prefLabel
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@en
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@nl
P2093
P2860
P1476
Cholesterol efflux potential a ...... nt with niacin or anacetrapib.
@en
P2093
Alan R Tall
Andrew K Taggart
Brian Hubbard
Carl P Sparrow
Jelena Kling
Tamara Pagler
Tim Fisher
P2860
P304
P356
10.1161/ATVBAHA.110.207142
P407
P577
2010-05-06T00:00:00Z