about
Human immunodeficiency virus type 1 and influenza virus exit via different membrane microdomains.HIV-1 assembly differentially alters dynamics and partitioning of tetraspanins and raft componentsGenetics of experimental allergic encephalomyelitis supports the role of T helper cells in multiple sclerosis pathogenesis.Histamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibilityIdentification of genetic determinants of the sexual dimorphism in CNS autoimmunity.Histamine H4 receptor optimizes T regulatory cell frequency and facilitates anti-inflammatory responses within the central nervous system.Gestational bisphenol A exposure and testis development.Exacerbation of autoimmune neuroinflammation by dietary sodium is genetically controlled and sex specific.The Lymphocytic Choriomeningitis Virus Matrix Protein PPXY Late Domain Drives the Production of Defective Interfering Particles.Inactivation of nuclear GSK3β by Ser(389) phosphorylation promotes lymphocyte fitness during DNA double-strand break responseStudies in experimental autoimmune encephalomyelitis do not support developmental bisphenol a exposure as an environmental factor in increasing multiple sclerosis risk.Environmental factors acting during development to influence MS risk: insights from animal studies.Natural genetic variation profoundly regulates gene expression in immune cells and dictates susceptibility to CNS autoimmunity.Sex-specific control of central nervous system autoimmunity by p38 mitogen-activated protein kinase signaling in myeloid cells.Histamine H₂ receptor signaling × environment interactions determine susceptibility to experimental allergic encephalomyelitis.The emerging role of p38 mitogen-activated protein kinase in multiple sclerosis and its models.Genetic variation in chromosome Y regulates susceptibility to influenza A virus infection.Tetraspanins regulate cell-to-cell transmission of HIV-1Anti-inflammatory roles of p38α MAPK in macrophages are context dependent and require IL-10.Modeling month-season of birth as a risk factor in mouse models of chronic disease: from multiple sclerosis to autoimmune encephalomyelitis.Vaccine draining lymph nodes are a source of antigen-specific B cells.Tuberculosis and impaired IL-23-dependent IFN-γ immunity in humans homozygous for a common missense variantCritical Role for SLAM/SAP Signaling in the Thymic Developmental Programming of IL-17- and IFN-γ-Producing γδ T CellsSex-Specific Gene-by-Vitamin D Interactions Regulate Susceptibility to Central Nervous System AutoimmunityNetwork-Based Functional Prediction Augments Genetic Association To Predict Candidate Genes for Histamine Hypersensitivity in Mice
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description
hulumtues
@sq
onderzoeker
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researcher
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հետազոտող
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name
Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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type
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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Dimitry N. Krementsov
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P106
P1153
6504667173
P21
P31
P496
0000-0003-2709-7466