Combination of sulindac and antimicrobial eradication of Helicobacter pylori prevents progression of gastric cancer in hypergastrinemic INS-GAS mice. - Wikidata - wikimedia - TriplyDB

Combination of sulindac and antimicrobial eradication of Helicobacter pylori prevents progression of gastric cancer in hypergastrinemic INS-GAS mice.

Combination of sulindac and antimicrobial eradication of Helicobacter pylori prevents progression of gastric cancer in hypergastrinemic INS-GAS mice.

http://www.wikidata.org/entity/Q42591340

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Effect of Helicobacter pylori Infection on the Composition of Gastric Microbiota in the Development of Gastric Cancer The gastrointestinal tumor microenvironment The human gastric microbiota: Is it time to rethink the pathogenesis of stomach diseases?Participation of microbiota in the development of gastric cancer Gastric colonisation with a restricted commensal microbiota replicates the promotion of neoplastic lesions by diverse intestinal microbiota in the Helicobacter pylori INS-GAS mouse model of gastric carcinogenesis Helminth co-infection in Helicobacter pylori infected INS-GAS mice attenuates gastric premalignant lesions of epithelial dysplasia and glandular atrophy and preserves colonization resistance of the stomach to lower bowel microbiota Alteration of stomach microbiota compositions in the progression of gastritis induces nitric oxide in gastric cell.Mouse models of gastric carcinogenesis.Lack of commensal flora in Helicobacter pylori-infected INS-GAS mice reduces gastritis and delays intraepithelial neoplasia.Apoptosis signal-regulating kinase 1 and cyclin D1 compose a positive feedback loop contributing to tumor growth in gastric cancer.The role of the gastrointestinal microbiome in Helicobacter pylori pathogenesis Systemic activation of K-ras rapidly induces gastric hyperplasia and metaplasia in mice Streptococcus mitis induces conversion of Helicobacter pylori to coccoid cells during co-culture in vitro.Helicobacter pylori Infection Induces Anemia, Depletes Serum Iron Storage, and Alters Local Iron-Related and Adult Brain Gene Expression in Male INS-GAS Mice.Bacterial overgrowth and diversification of microbiota in gastric cancer Imbalance of Gastrointestinal Microbiota in the Pathogenesis of Helicobacter pylori-Associated Diseases.Rodent models of Helicobacter infection, inflammation, and disease Mouse models of gastric cancer.Mouse models for gastric cancer: Matching models to biological questions.Mucosal microbiome dysbiosis in gastric carcinogenesis.Impact of the Microbiota and Gastric Disease Development by Helicobacter pylori.Antibacterial effects of the urushiol component in the sap of the lacquer tree (Rhus verniciflua Stokes) on Helicobacter pylori.The gastric microbiome, its interaction with Helicobacter pylori, and its potential role in the progression to stomach cancer.

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P2860

Combination of sulindac and antimicrobial eradication of Helicobacter pylori prevents progression of gastric cancer in hypergastrinemic INS-GAS mice.

http://www.wikidata.org/entity/Q42591340

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Peiying Yang

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Timothy C Wang

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P2860

Nonsteroidal Anti-inflammatory Drugs and Cyclooxygenase-2 Inhibitors for Primary Prevention of Colorectal Cancer: A Systematic Review Prepared for the U.S. Preventive Services Task Force

Current concepts in the management of Helicobacter pylori infection: the Maastricht III Consensus Report

Inflammation, atrophy, and gastric cancer

Non-steroidal anti-inflammatory drug use and the risk of gastric cancer: a systematic review and meta-analysis

Unidentified curved bacilli in the stomach of patients with gastritis and peptic ulceration

The effect of celecoxib, a cyclooxygenase-2 inhibitor, in familial adenomatous polyposis

Spatial distribution and stability of the eight microbial species of the altered schaedler flora in the mouse gastrointestinal tract

Effects of nonsteroidal anti-inflammatory drugs on Helicobacter pylori-infected gastric mucosae of mice: apoptosis, cell proliferation, and inflammatory activity.

Helicobacter pylori related hypergastrinaemia is the result of a selective increase in gastrin 17

Sulindac causes regression of rectal polyps in familial adenomatous polyposis.

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eradication of infectious diseases

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