Dot1 binding induces chromatin rearrangements by histone methylation-dependent and -independent mechanisms.
about
The role of multiple marks in epigenetic silencing and the emergence of a stable bivalent chromatin stateNormally lethal amino acid substitutions suppress an ultramutator DNA Polymerase δ variantEpigenetics in Saccharomyces cerevisiae.Chromatin-modifying enzymes as modulators of reprogrammingEpigenetic Editing: targeted rewriting of epigenetic marks to modulate expression of selected target genes.Emergence of DNA polymerase ε antimutators that escape error-induced extinction in yeast.The histone methyltransferase Dot1/DOT1L as a critical regulator of the cell cycle.Getting down to the core of histone modifications.Transcriptional activator DOT1L putatively regulates human embryonic stem cell differentiation into the cardiac lineage.TERRA and the histone methyltransferase Dot1 cooperate to regulate senescence in budding yeast.
P2860
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P2860
Dot1 binding induces chromatin rearrangements by histone methylation-dependent and -independent mechanisms.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Dot1 binding induces chromatin ...... t and -independent mechanisms.
@en
Dot1 binding induces chromatin ...... t and -independent mechanisms.
@nl
type
label
Dot1 binding induces chromatin ...... t and -independent mechanisms.
@en
Dot1 binding induces chromatin ...... t and -independent mechanisms.
@nl
prefLabel
Dot1 binding induces chromatin ...... t and -independent mechanisms.
@en
Dot1 binding induces chromatin ...... t and -independent mechanisms.
@nl
P2093
P2860
P50
P356
P1476
Dot1 binding induces chromatin ...... nt and -independent mechanisms
@en
P2093
Alex W Faber
Floor Frederiks
Frank Cp Holstege
Iris Je Stulemeijer
Kitty F Verzijlbergen
Tibor van Welsem
P2860
P2888
P356
10.1186/1756-8935-4-2
P577
2011-02-03T00:00:00Z
P5875
P6179
1050124085