Pancreatic digestive enzyme blockade in the intestine increases survival after experimental shock.
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Redefining the gut as the motor of critical illnessThe digestive tract as the origin of systemic inflammationTransmural intestinal wall permeability in severe ischemia after enteral protease inhibition.Mechanisms by which Stress Affects the Experimental and Clinical Inflammatory Bowel Disease (IBD): Role of Brain-Gut Axis.In vivo analysis of intestinal permeability following hemorrhagic shockShockOmics: multiscale approach to the identification of molecular biomarkers in acute heart failure induced by shock.Peptidomic Analysis of Rat Plasma: Proteolysis in Hemorrhagic ShockProteolytic receptor cleavage in the pathogenesis of blood rheology and co-morbidities in metabolic syndrome. Early forms of autodigestion.Removal of luminal content protects the small intestine during hemorrhagic shock but is not sufficient to prevent lung injury.The autodigestion hypothesis for shock and multi-organ failure.Autodigestion: Proteolytic Degradation and Multiple Organ Failure in ShockIntraluminal tranexamic acid inhibits intestinal sheddases and mitigates gut and lung injury and inflammation in a rodent model of hemorrhagic shock.Regional susceptibility to stress-induced intestinal injury in the mouse.The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.Pancreatic digestive enzyme blockade in the small intestine prevents insulin resistance in hemorrhagic shock.Volatile Decay Products in Breath During Peritonitis Shock are Attenuated by Enteral Blockade of Pancreatic Digestive Proteases.Intraluminal nonbacterial intestinal components control gut and lung injury after trauma hemorrhagic shock.Is Matrix Metalloproteinase-8 Activity in the Mucosal Barrier a Requirement for Leakage of Cecal Material in Peritonitis?Enteral tranexamic acid attenuates vasopressor resistance and changes in α1-adrenergic receptor expression in hemorrhagic shock.Hemorrhagic Shock and the Microvasculature.Innate immune responses to trauma.Re: ADAM-17: A potential therapeutic target to prevent organ injury after hemorrhagic shock?
P2860
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P2860
Pancreatic digestive enzyme blockade in the intestine increases survival after experimental shock.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@en
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@nl
type
label
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@en
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@nl
prefLabel
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@en
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@nl
P2093
P2860
P1476
Pancreatic digestive enzyme bl ...... ival after experimental shock.
@en
P2093
David B Hoyt
Frank A DeLano
Geert W Schmid-Schönbein
P2860
P304
P356
10.1126/SCITRANSLMED.3005046
P407
P577
2013-01-01T00:00:00Z