Vagal nerve stimulation protects against burn-induced intestinal injury through activation of enteric glia cells.
about
Parasympathetic stimulation via the vagus nerve prevents systemic organ dysfunction by abrogating gut injury and lymph toxicity in trauma and hemorrhagic shockRole of enteric neurotransmission in host defense and protection of the gastrointestinal tractVagus nerve stimulation: from epilepsy to the cholinergic anti-inflammatory pathwayCHRFAM7A, a human-specific and partially duplicated α7-nicotinic acetylcholine receptor gene with the potential to specify a human-specific inflammatory response to injuryClostridial toxins: sensing a target in a hostile gut environment.The vagus nerve in appetite regulation, mood and intestinal inflammationHomeostasis of the gut barrier and potential biomarkers.Enteric glial cells and their role in the intestinal epithelial barrierPostinjury vagal nerve stimulation protects against intestinal epithelial barrier breakdownIntravenous phage display identifies peptide sequences that target the burn-injured intestine.Efferent vagal nerve stimulation attenuates acute lung injury following burn: The importance of the gut-lung axisImportance of NO and its related compounds in enteric nervous system regulation of gut homeostasis and disease susceptibilityS-nitrosothiol signals in the enteric nervous system: lessons learnt from big brother.Enteric glia cells attenuate cytomix-induced intestinal epithelial barrier breakdown.Electroacupuncture activates enteric glial cells and protects the gut barrier in hemorrhaged ratsA Human-Specific α7-Nicotinic Acetylcholine Receptor Gene in Human Leukocytes: Identification, Regulation and the Consequences of CHRFAM7A Expression.Vagal nerve stimulation blocks peritoneal macrophage inflammatory responsiveness after severe burn injuryModulating the Biologic Activity of Mesenteric Lymph after Traumatic Shock Decreases Systemic Inflammation and End Organ Injury.Enteric Glial Cells: A New Frontier in Neurogastroenterology and Clinical Target for Inflammatory Bowel Diseases.Electroacupuncture at Zusanli (ST36) Prevents Intestinal Barrier and Remote Organ Dysfunction following Gut Ischemia through Activating the Cholinergic Anti-Inflammatory-Dependent MechanismRole of non-muscle myosin light chain kinase in neutrophil-mediated intestinal barrier dysfunction during thermal injuryFetal cholinergic anti-inflammatory pathway and necrotizing enterocolitis: the brain-gut connection begins in uteroElectroacupuncture improves gut barrier dysfunction in prolonged hemorrhagic shock rats through vagus anti-inflammatory mechanism.Novel functional roles for enteric glia in the gastrointestinal tract.The digestive neuronal-glial-epithelial unit: a new actor in gut health and disease.The past 10 years of gastroenterology and hepatology-reflections and predictions.Plasticity of Schwann cells and pericytes in response to islet injury in mice.Non-alcoholic fatty liver disease as a consequence of autonomic imbalance and circadian desynchronization.CHRFAM7A: a human-specific α7-nicotinic acetylcholine receptor gene shows differential responsiveness of human intestinal epithelial cells to LPSGastric electrical stimulation improves enteric neuronal survivalSacral nerve stimulation enhances early intestinal mucosal repair following mucosal injury in a pig model.Potential roles of enteric glia in bridging neuroimmune communication in the gut.Vagus nerve stimulation: a new promising therapeutic tool in inflammatory bowel disease.Neuroimmune Modulation of Gut Function.Vagal nerve stimulation modulates gut injury and lung permeability in trauma-hemorrhagic shock.CPSI-121 pharmacologically prevents intestinal barrier dysfunction after cutaneous burn through a vagus nerve-dependent mechanism.Vagal nerve stimulation protects cardiac injury by attenuating mitochondrial dysfunction in a murine burn injury model.Vagus nerve stimulation blocks vascular permeability following burn in both local and distal sites.Effects of α-conotoxin ImI on TNF-α, IL-8 and TGF-β expression by human macrophage-like cells derived from THP-1 pre-monocytic leukemic cellsEnteric glia cells are critical to limiting the intestinal inflammatory response after injury.
P2860
Q24632596-EFF86D73-3882-4F89-975E-05A56125E4E5Q26827692-4326106B-43AC-4678-90FB-382D09F0C9D4Q26858806-B2C215B5-55BF-43A9-9578-F41192BB39EEQ26996489-5753AB84-8F74-4517-AB56-E5BC7197CBB8Q27016565-2E9F147C-3E6C-4EC1-AD48-5B3F98C77973Q28070016-21C4953A-BB68-494F-9688-FB7D22FDEA6EQ33715804-AD9C6099-4ED5-4614-968A-B7271AD33FA7Q34093834-6419D205-332C-445D-9469-8D507B9BC647Q34187168-12EE9ADD-D1D0-41E7-BC31-DFC9D15B7F86Q34409138-13FE86CE-2F68-4946-B247-0ED8E3C047D4Q34609771-CD11D497-65F8-45FD-856E-CC626F210DE9Q34623553-BC45C56E-8913-4E22-A321-D90D3A4125D7Q34677895-EBBDAEBF-CE63-4731-BFE7-823CCCD6F775Q34809293-EC21DE64-6374-4B18-8388-ABAC49411F0DQ35045716-9F21C9DB-321D-46F7-9391-8B7A38DA5FF4Q35946779-A7F4BAD2-7309-4C8D-B3B3-79E28B4753D7Q36173793-D1CE14E2-1A13-4A5A-A5F9-CB7837BEB312Q36225261-7E06F7A5-3480-48D7-B230-5367A2CA2B43Q36477852-863CDCB0-E214-4394-9F12-878E1677283AQ36801752-C2AD72EE-664B-413C-9B34-E37AC91E59F7Q37026888-B877CA7D-D8EC-4147-9103-0FE17A416C32Q37080684-D23C5882-4FF2-4BF7-9727-964A4F19F827Q37203535-1EAFB090-E37F-4B6D-B7B5-BAFAAD6ED7C1Q38034376-0FDB1EDC-5331-4012-86B6-AA8BD575A741Q38060991-A792EEA9-2038-43C0-AE2D-D6B8F089991EQ38257761-7845928C-FBBA-4BE4-AE58-CC031CFAC699Q38425691-54C39C5D-5D97-43B6-AA6D-3AB713797EF6Q38555120-55E2AABC-BCB9-4219-BE7E-C373226EEA9DQ38598715-264D243C-6D66-46A8-A52E-51A8FAF97913Q38699909-D8C1FCFF-3359-4CE5-A478-907AE9473C17Q38900423-457BE055-B859-4A68-93D8-9C091567F876Q39049151-9FA0B145-E2CA-4510-B10D-03926B5D11DEQ39250092-F0BE021A-E695-483D-931B-5493B14826C7Q40431964-7AC99C04-0A52-45CB-82A9-517C4A5A05A7Q41952215-51D2A73B-D316-49E5-B2BC-3F285A6C054EQ41972904-DC3244B5-A35B-4455-8A4B-D901271D467BQ42051538-A1958547-9B2E-4806-93CC-9B9B689E6646Q42325585-36E3CAC7-F699-490F-AA23-9C6E21D6E2D7Q42371770-A60E7B52-C53F-48EA-91CE-90071A789701Q46146227-AE4BA697-E6CC-49BD-9F7D-B3EB374D4A86
P2860
Vagal nerve stimulation protects against burn-induced intestinal injury through activation of enteric glia cells.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en-gb
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@nl
type
label
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en-gb
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@nl
prefLabel
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en-gb
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@nl
P2093
P2860
P356
P1476
Vagal nerve stimulation protec ...... ivation of enteric glia cells.
@en
P2093
Carrie Y Peterson
James G Putnam
Michael Krzyzaniak
Raul Coimbra
Todd W Costantini
Vishal Bansal
William H Loomis
P2860
P304
P356
10.1152/AJPGI.00156.2010
P577
2010-08-12T00:00:00Z