Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
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TRP Channels in Respiratory Pathophysiology: the Role of Oxidative, Chemical Irritant and Temperature StimuliDual regulation of diacylglycerol kinase (DGK)-θ: polybasic proteins promote activation by phospholipids and increase substrate affinityThe Role of Transient Receptor Potential Channel 6 Channels in the Pulmonary VasculatureClassical transient receptor potential 1 and 6 contribute to hypoxic pulmonary hypertension through differential regulation of pulmonary vascular functions.CFTR and sphingolipids mediate hypoxic pulmonary vasoconstrictionDiscovery and pharmacological characterization of a novel potent inhibitor of diacylglycerol-sensitive TRPC cation channelsTransient receptor potential channels in the vasculature.Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension.Gain-of-function mutations in transient receptor potential C6 (TRPC6) activate extracellular signal-regulated kinases 1/2 (ERK1/2)Gap junctions support the sustained phase of hypoxic pulmonary vasoconstriction by facilitating calcium sensitization.Optimization of isolated perfused/ventilated mouse lung to study hypoxic pulmonary vasoconstriction.Hypoxia-dependent reactive oxygen species signaling in the pulmonary circulation: focus on ion channels.Lungs at high-altitude: genomic insights into hypoxic responses.Role of phosphatase and tensin homolog in hypoxic pulmonary vasoconstriction.Diacylglycerol kinase θ: regulation and stability.Pharmacological inhibition of FSGS-related TRPC6 gain of function mutants by semisynthetic larixol-derived compounds.Important role of PLC-γ1 in hypoxic increase in intracellular calcium in pulmonary arterial smooth muscle cells.The fatty acid composition of diacylglycerols determines local signaling patterns.Recent advances in oxygen sensing and signal transduction in hypoxic pulmonary vasoconstriction.Choline Triggers Exacerbations of Chronic Obstructive Pulmonary Disease in Patients Infected with Pseudomonas aeruginosa.Hypoxia Selectively Upregulates Cation Channels and Increases Cytosolic [Ca2+] in Pulmonary, but not Coronary, Arterial Smooth Muscle Cells.The endothelium in hypoxic pulmonary vasoconstriction.An optically controlled probe identifies lipid-gating fenestrations within the TRPC3 channel.Integrative understanding of hypoxic pulmonary vasoconstriction using in vitro models: from ventilated/perfused lung to single arterial myocyte.
P2860
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P2860
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
@en
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
@nl
type
label
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
@en
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
@nl
prefLabel
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
@en
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.
@nl
P2093
P2860
P50
P356
P1433
P1476
Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6
@en
P2093
Beate Fuchs
Markus Rupp
Thomas Gudermann
P2860
P2888
P356
10.1186/1465-9921-12-20
P50
P577
2011-02-04T00:00:00Z
P5875
P6179
1031974940