Inhibition of 11beta-hydroxysteroid dehydrogenase, the foeto-placental barrier to maternal glucocorticoids, permanently programs amygdala GR mRNA expression and anxiety-like behaviour in the offspring.
about
Natural variation in gestational cortisol is associated with patterns of growth in marmoset monkeys (Callithrix geoffroyi)PTSD and gene variants: new pathways and new thinking.Minireview: the case for obesogensEndocrine disrupters as obesogensFetal programming and early identification of newborns at high risk of free radical-mediated diseasesPrenatal excess glucocorticoid exposure and adult affective disorders: a role for serotonergic and catecholamine pathwaysBrain mineralocorticoid receptors in cognition and cardiovascular homeostasisFetal stress and programming of hypoxic/ischemic-sensitive phenotype in the neonatal brain: mechanisms and possible interventionsCord Blood DNA Methylation Biomarkers for Predicting Neurodevelopmental OutcomesModulation of the Hypothalamic-Pituitary-Adrenal Axis by Early Life Stress ExposurePrenatal stress: role in psychotic and depressive diseases.Context modulates outcome of perinatal glucocorticoid action in the brain.The effect of stress and anxiety associated with maternal prenatal diagnosis on feto-maternal attachment.Stressed-out, or in (utero)?Feed allowance and maternal backfat levels during gestation influence maternal cortisol levels, milk fat composition and offspring growth.Reduction of hepatic glucocorticoid receptor and hexose-6-phosphate dehydrogenase expression ameliorates diet-induced obesity and insulin resistance in mice.Placental 11-beta hydroxysteroid dehydrogenase methylation is associated with newborn growth and a measure of neurobehavioral outcome.Epigenetic signaling in psychiatric disorders.Epigenetic effects of prenatal stress on 11β-hydroxysteroid dehydrogenase-2 in the placenta and fetal brainConsequences of serotonin transporter genotype and early adversity on behavioral profile - pathology or adaptation?Epigenetic signaling in psychiatric disorders: stress and depression.Maternal stress induces epigenetic signatures of psychiatric and neurological diseases in the offspringEarly onset pre-eclampsia is associated with altered DNA methylation of cortisol-signalling and steroidogenic genes in the placentaJuvenile offspring of rats exposed to restraint stress in late gestation have impaired cognitive performance and dysregulated progestogen formation.Prenatal exposure to glucocorticoids and adult disease.11β-hydroxysteroid dehydrogenases and the brain: from zero to hero, a decade of progress.Maternal depression and anxiety are associated with altered gene expression in the human placenta without modification by antidepressant use: implications for fetal programmingFetal origin of vascular aging.Prenatal dexamethasone exposure induces changes in nonhuman primate offspring cardiometabolic and hypothalamic-pituitary-adrenal axis function.Programming of the hypothalamo-pituitary-adrenal axis: serotonergic involvement.Fetal brain 11β-hydroxysteroid dehydrogenase type 2 selectively determines programming of adult depressive-like behaviors and cognitive function, but not anxiety behaviors in male mice.Obesogens, stem cells and the developmental programming of obesity.The secret language of destiny: stress imprinting and transgenerational origins of diseaseGestational exposure to variable stressors produces decrements in cognitive and neural development of juvenile male and female rats.Fetal origins of adult disease.The Role of Placental 11-Beta Hydroxysteroid Dehydrogenase Type 1 and Type 2 Methylation on Gene Expression and Infant Birth Weight.Physiological reactivity to psychological stress in human pregnancy: current knowledge and future directionsPlacental adaptive responses and fetal programming.The limited effects of obstetrical and neonatal complications on conduct and attention-deficit hyperactivity disorder symptoms in middle childhood.Glucocorticoid "programming" and PTSD risk.
P2860
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P2860
Inhibition of 11beta-hydroxysteroid dehydrogenase, the foeto-placental barrier to maternal glucocorticoids, permanently programs amygdala GR mRNA expression and anxiety-like behaviour in the offspring.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@en
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@nl
type
label
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@en
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@nl
prefLabel
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@en
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@nl
P2093
P1476
Inhibition of 11beta-hydroxyst ...... ke behaviour in the offspring.
@en
P2093
P304
P356
10.1046/J.1460-9568.2000.00958.X
P407
P577
2000-03-01T00:00:00Z