Late-onset Alzheimer's disease is associated with inherent changes in bioenergetics profiles.

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Ursodeoxycholic Acid Improves Mitochondrial Function and Redistributes Drp1 in Fibroblasts from Patients with Either Sporadic or Familial Alzheimer's Disease

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Q57479995-F12328C3-A305-4FEF-B71B-ABAC1312F7D2

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Late-onset Alzheimer's disease is associated with inherent changes in bioenergetics profiles.

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http://www.wikidata.org/entity/Q42630551

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

@zh-hk

2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

Late-onset Alzheimer's disease ...... ges in bioenergetics profiles.

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Late-onset Alzheimer's disease ...... ges in bioenergetics profiles.

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Item

label

Late-onset Alzheimer's disease ...... ges in bioenergetics profiles.

@en

Late-onset Alzheimer's disease ...... ges in bioenergetics profiles.

@nl

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Late-onset Alzheimer's disease ...... ges in bioenergetics profiles.

@en

Late-onset Alzheimer's disease ...... ges in bioenergetics profiles.

@nl

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Late-onset Alzheimer's disease ...... nges in bioenergetics profiles

@en

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Arthur J Siegel

http://www.w3.org/2001/XMLSchema#string

Brent Forester

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Bruce M Cohen

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Donna L McPhie

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Kristopher M Amirault

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Ryan A Healy

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Woo-In Ryu

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Amyloid-β and Astrocytes Interplay in Amyloid-β Related Disorders

Metabolomics of human brain aging and age-related neurodegenerative diseases

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

6-Phosphofructo-2-kinase (PFKFB3) promotes cell cycle progression and suppresses apoptosis via Cdk1-mediated phosphorylation of p27

THE METABOLISM OF TUMORS IN THE BODY

Decreased clearance of CNS beta-amyloid in Alzheimer's disease

Neurovascular regulation in the normal brain and in Alzheimer's disease

Membrane potential can be determined in individual cells from the nernstian distribution of cationic dyes.

Overexpression of pyruvate dehydrogenase kinase 1 and lactate dehydrogenase A in nerve cells confers resistance to amyloid β and other toxins by decreasing mitochondrial respiration and reactive oxygen species production.

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s41598-017-14420-x

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14038

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scholarly article

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10.1038/S41598-017-14420-X

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English

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http://www.w3.org/2001/XMLSchema#string

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http://www.w3.org/2001/XMLSchema#string

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Kai-Christian Sonntag

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2017-10-25T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

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29070876

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Alzheimer's disease

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5656579

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Late-onset Alzheimer's disease is associated with inherent changes in bioenergetics profiles.

about

P2860

P2860

Late-onset Alzheimer's disease is associated with inherent changes in bioenergetics profiles.

description

name

type

label

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P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P50

P577

P698

P921

P932