about
Aberrant RNA splicing and its functional consequences in cancer cellsBRCA1: cell cycle checkpoint, genetic instability, DNA damage response and cancer evolutionEvolutionary conservation analysis increases the colocalization of predicted exonic splicing enhancers in the BRCA1 gene with missense sequence changes and in-frame deletions, but not polymorphismsThe role of BRCA1-IRIS in the development and progression of triple negative breast cancers in Egypt: possible link to disease early lesionBRCA1-IRIS overexpression promotes formation of aggressive breast cancers.BRCA1 pathway function in basal-like breast cancer cells.The breast cancer susceptibility genes (BRCA) in breast and ovarian cancers.Induction of breast cancer in wild type p53 cells by BRCA1-IRIS overexpression.BRCA1-IRIS inactivation overcomes paclitaxel resistance in triple negative breast cancers.Hyperplasia and spontaneous tumor development in the gynecologic system in mice lacking the BRCA1-Delta11 isoform.Geminin overexpression promotes imatinib sensitive breast cancer: a novel treatment approach for aggressive breast cancers, including a subset of triple negativeGeminin overexpression prevents the completion of topoisomerase IIα chromosome decatenation, leading to aneuploidy in human mammary epithelial cellsBRCA1/p220 loss triggers BRCA1-IRIS overexpression via mRNA stabilization in breast cancer cellsAKT regulates BRCA1 stability in response to hormone signaling.Regulation of the Wip1 phosphatase and its effects on the stress response.Mouse models of BRCA1 and BRCA2 deficiency: past lessons, current understanding and future prospects.Mitochondrial localization, ELK-1 transcriptional regulation and growth inhibitory functions of BRCA1, BRCA1a, and BRCA1b proteinsThe role of BRCA1-IRIS in ovarian cancer formation, drug resistance and progression.Notch-1 activates estrogen receptor-alpha-dependent transcription via IKKalpha in breast cancer cells.Preclinical mouse models for BRCA1-associated breast cancer.Impaired skin and mammary gland development and increased gamma-irradiation-induced tumorigenesis in mice carrying a mutation of S1152-ATM phosphorylation site in Brca1.BRCA1-IRIS overexpression promotes and maintains the tumor initiating phenotype: implications for triple negative breast cancer early lesions.BRCA1 exon 11 alternative splicing, multiple functions and the association with cancer.Mouse models of BRCA1 and their application to breast cancer research.VHR/DUSP3 phosphatase: structure, function and regulation.BRCA1-IRIS inactivation sensitizes ovarian tumors to cisplatin.Geminin overexpression-dependent recruitment and crosstalk with mesenchymal stem cells enhance aggressiveness in triple negative breast cancers.Comprehensive annotation of splice junctions supports pervasive alternative splicing at the BRCA1 locus: a report from the ENIGMA consortium.A niche that triggers aggressiveness within BRCA1-IRIS overexpressing triple negative tumors is supported by reciprocal interactions with the microenvironment.Detecting splicing patterns in genes involved in hereditary breast and ovarian cancer.Promoter usage of BRCA1-IRIS.Premature polyadenylation of MAGI3 is associated with diminished N6-methyladenosine in its large internal exon.Revisiting the roles of VHR/DUSP3 phosphatase in human diseasesUbiquitin ligase RNF8 suppresses Notch signaling to regulate mammary development and tumorigenesis
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Identification of BRCA1-IRIS, a BRCA1 locus product.
@en
Identification of BRCA1-IRIS, a BRCA1 locus product.
@nl
type
label
Identification of BRCA1-IRIS, a BRCA1 locus product.
@en
Identification of BRCA1-IRIS, a BRCA1 locus product.
@nl
prefLabel
Identification of BRCA1-IRIS, a BRCA1 locus product.
@en
Identification of BRCA1-IRIS, a BRCA1 locus product.
@nl
P2860
P356
P1433
P1476
Identification of BRCA1-IRIS, a BRCA1 locus product.
@en
P2093
David M Livingston
Wael M ElShamy
P2860
P2888
P304
P356
10.1038/NCB1171
P577
2004-09-26T00:00:00Z