Altered fracture repair in the absence of MMP9 - Wikidata - wikimedia - TriplyDB

Altered fracture repair in the absence of MMP9

Altered fracture repair in the absence of MMP9

http://www.wikidata.org/entity/Q42641175

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Lead exposure inhibits fracture healing and is associated with increased chondrogenesis, delay in cartilage mineralization, and a decrease in osteoprogenitor frequency.Overview of biological mechanisms and applications of three murine models of bone repair: closed fracture with intramedullary fixation, distraction osteogenesis, and marrow ablation by reaming Unraveling macrophage contributions to bone repair Current insights on the regenerative potential of the periosteum: molecular, cellular, and endogenous engineering approaches Role of matrix metalloproteinase 13 in both endochondral and intramembranous ossification during skeletal regeneration Osteopotentia regulates osteoblast maturation, bone formation, and skeletal integrity in mice Galectin-3 is a downstream regulator of matrix metalloproteinase-9 function during endochondral bone formation.Tendon regeneration and scar formation: The concept of scarless healing Matrix metalloproteinases and the regulation of tissue remodelling Angiogenesis in bone regeneration.Accelerated bone repair after plasma laser corticotomies.Identification of candidate angiogenic inhibitors processed by matrix metalloproteinase 2 (MMP-2) in cell-based proteomic screens: disruption of vascular endothelial growth factor (VEGF)/heparin affin regulatory peptide (pleiotrophin) and VEGF/Conne Salivary proteomics in bisphosphonate-related osteonecrosis of the jaw.A finite element inverse analysis to assess functional improvement during the fracture healing process Cellular basis for age-related changes in fracture repair Mepe is expressed during skeletal development and regeneration.Effect of bone morphogenetic protein signaling on development of the jaw skeleton.Influence of heating and cyclic tension on the induction of heat shock proteins and bone-related proteins by MC3T3-E1 cells.Genetic association of angiogenesis- and hypoxia-related gene polymorphisms with osteonecrosis of the femoral head Interaction of age and mechanical stability on bone defect healing: an early transcriptional analysis of fracture hematoma in rat Site specific effects of zoledronic acid during tibial and mandibular fracture repair.Microarray Analysis of Gene Expression Reveals that Cyclo-oxygenase-2 Gene Therapy Up-regulates Hematopoiesis and Down-regulates Inflammation During Endochondral Bone Fracture Healing.Remodeling of cortical bone allografts mediated by adherent rAAV-RANKL and VEGF gene therapy.Matrix metalloproteinase-27 is expressed in CD163+/CD206+ M2 macrophages in the cycling human endometrium and in superficial endometriotic lesions.Identification of CITED2 as a negative regulator of fracture healing.Molecular analysis of healing at a bone-implant interface.The biology of fracture healing.Molecular mechanisms controlling bone formation during fracture healing and distraction osteogenesis Matrix metalloproteinase activity and osteoclasts in experimental prostate cancer bone metastasis tissue.Acute phosphate restriction leads to impaired fracture healing and resistance to BMP-2.A surgical procedure for resecting the mouse rib: a model for large-scale long bone repair.Mechanical stability affects angiogenesis during early fracture healing.Loss of transcription factor early growth response gene 1 results in impaired endochondral bone repair.Urine matrix metalloproteinases (MMPs) as biomarkers for the progression of fracture healing Impaired Fracture Healing Caused by Deficiency of the Immunoreceptor Adaptor Protein DAP12.Matrix-metalloproteinase-9 is cleaved and activated by cathepsin K.Generation of closed transverse fractures in small animals.Matrix metalloproteinase-dependent microsomal prostaglandin E synthase-1 expression in macrophages: role of TNF-α and the EP4 prostanoid receptor.Matrix remodeling during endochondral ossification.Differential effects between the loss of MMP-2 and MMP-9 on structural and tissue-level properties of bone.

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P2860

Altered fracture repair in the absence of MMP9

http://www.wikidata.org/entity/Q42641175

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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2003年论文

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2003年论文

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name

Altered fracture repair in the absence of MMP9

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Altered fracture repair in the absence of MMP9

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type

label

Altered fracture repair in the absence of MMP9

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Altered fracture repair in the absence of MMP9

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prefLabel

Altered fracture repair in the absence of MMP9

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Altered fracture repair in the absence of MMP9

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Altered fracture repair in the absence of MMP9

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Céline Colnot

http://www.w3.org/2001/XMLSchema#string

Jill A Helms

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Theodore Miclau

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Zachary Thompson

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Zena Werb

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P2860

MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes

Recruitment of stem and progenitor cells from the bone marrow niche requires MMP-9 mediated release of kit-ligand

Matrix metalloproteinase-9 triggers the angiogenic switch during carcinogenesis

Mutation of the matrix metalloproteinase 2 gene (MMP2) causes a multicentric osteolysis and arthritis syndrome

Leakage-resistant blood vessels in mice transgenically overexpressing angiopoietin-1

Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18

Mechanisms of angiogenesis

Clinically applied models of bone regeneration in tissue engineering research.

Vascular endothelial growth factor stimulates bone repair by promoting angiogenesis and bone turnover.

HIF-1: using two hands to flip the angiogenic switch.

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10.1242/DEV.00559

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17

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