The complete sequence of the locus of enterocyte effacement (LEE) from enteropathogenic Escherichia coli E2348/69.
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Bacteria-host communication: the language of hormonesBioinformatics analysis of the locus for enterocyte effacement provides novel insights into type-III secretionFrom ingestion to colonization: the influence of the host environment on regulation of the LEE encoded type III secretion system in enterohaemorrhagic Escherichia coliEscherichia coli type III secretion system 2: a new kind of T3SS?Tyrosine-phosphorylated caveolin-1 blocks bacterial uptake by inducing Vav2-RhoA-mediated cytoskeletal rearrangementsType Three Secretion System in Attaching and Effacing PathogensTranslocated EspF protein from enteropathogenic Escherichia coli disrupts host intestinal barrier functionYscO of Yersinia pestis is a mobile core component of the Yop secretion systemA Highly Conserved Bacterial D-Serine Uptake System Links Host Metabolism and VirulenceGenes encoding putative effector proteins of the type III secretion system of Salmonella pathogenicity island 2 are required for bacterial virulence and proliferation in macrophagesEnteric Pathogens Exploit the Microbiota-generated Nutritional Environment of the GutBacterial Adrenergic Sensors Regulate Virulence of Enteric Pathogens in the GutNck adaptors, besides promoting N-WASP mediated actin-nucleation activity at pedestals, influence the cellular levels of enteropathogenic Escherichia coli Tir effector.A novel two-component signaling system that activates transcription of an enterohemorrhagic Escherichia coli effector involved in remodeling of host actin.Cortactin is essential for F-actin assembly in enteropathogenic Escherichia coli (EPEC)- and enterohaemorrhagic E. coli (EHEC)-induced pedestals and the alpha-helical region is involved in the localization of cortactin to bacterial attachment sites.Role of EspF in host cell death induced by enteropathogenic Escherichia coli.Roles of the intestinal microbiota in pathogen protection.Genomic diversity of EPEC associated with clinical presentations of differing severity.Evolution of atypical enteropathogenic E. coli by repeated acquisition of LEE pathogenicity island variants.The Type Three Secretion System 2-Encoded Regulator EtrB Modulates Enterohemorrhagic Escherichia coli Virulence Gene Expression.Comparative Genomics Provides Insight into the Diversity of the Attaching and Effacing Escherichia coli Virulence Plasmids.Immunogenic Domains and Secondary Structure of Escherichia coli Recombinant Secreted Protein Escherichia coli-Secreted Protein BHigh-throughput microfluidic method to study biofilm formation and host-pathogen interactions in pathogenic Escherichia coli.An activator of glutamate decarboxylase genes regulates the expression of enteropathogenic Escherichia coli virulence genes through control of the plasmid-encoded regulator, Per.Reactivity of convalescent-phase hemolytic-uremic syndrome patient sera with the megaplasmid-encoded TagA protein of Shiga toxigenic Escherichia coli O157Molecular characterization of the locus encoding biosynthesis of the lipopolysaccharide O antigen of Escherichia coli serotype O113A C-terminal class I PDZ binding motif of EspI/NleA modulates the virulence of attaching and effacing Escherichia coli and Citrobacter rodentium.The EspB protein of enteropathogenic Escherichia coli is targeted to the cytoplasm of infected HeLa cells.Involvement of the intermediate filament protein cytokeratin-18 in actin pedestal formation during EPEC infection.Comparative analysis of locus of enterocyte effacement pathogenicity islands of atypical enteropathogenic Escherichia coli.Transcriptional inhibitor of virulence factors in enteropathogenic Escherichia coli.Protection of rabbits against enteropathogenic Escherichia coli (EPEC) using an intimin null mutantVirulence factors of Escherichia coli O157 and other Shiga toxin-producing E. coli.Distribution of the secondary type III secretion system locus found in enterohemorrhagic Escherichia coli O157:H7 isolates among Shiga toxin-producing E. coli strains.RegA, an AraC-like protein, is a global transcriptional regulator that controls virulence gene expression in Citrobacter rodentium.Shiga toxin-producing Escherichia coli strains negative for locus of enterocyte effacementGlucose decreases virulence gene expression of Escherichia coli O157:H7.Enterohemorrhagic E. coli (EHEC) pathogenesis.Secretion of flagellin by the LEE-encoded type III secretion system of enteropathogenic Escherichia coli.Two distinct groups of porcine enteropathogenic Escherichia coli strains of serogroup O45 are revealed by comparative genomic hybridization and virulence gene microarray.
P2860
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P2860
The complete sequence of the locus of enterocyte effacement (LEE) from enteropathogenic Escherichia coli E2348/69.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
The complete sequence of the l ...... nic Escherichia coli E2348/69.
@en
The complete sequence of the locus of enterocyte effacement
@nl
type
label
The complete sequence of the l ...... nic Escherichia coli E2348/69.
@en
The complete sequence of the locus of enterocyte effacement
@nl
prefLabel
The complete sequence of the l ...... nic Escherichia coli E2348/69.
@en
The complete sequence of the locus of enterocyte effacement
@nl
P2093
P2860
P1476
The complete sequence of the l ...... enic Escherichia coli E2348/69
@en
P2093
B P McNamara
K G Jarvis
L A Wainwright
S J Elliott
T K McDaniel
P2860
P356
10.1046/J.1365-2958.1998.00783.X
P407
P577
1998-04-01T00:00:00Z