Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
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Blocking neutrophil diapedesis prevents hemorrhage during thrombocytopenia.Gαi2- and Gαi3-deficient mice display opposite severity of myocardial ischemia reperfusion injuryInsulin secretion stimulated by L-arginine and its metabolite L-ornithine depends on Gα(i2)Gαi1 and Gαi3 regulate macrophage polarization by forming a complex containing CD14 and Gab1.Platelet Gi protein Gαi2 is an essential mediator of thrombo-inflammatory organ damage in mice.Bronchial lesions of mouse model of asthma are preceded by immune complex vasculitis and induced bronchial associated lymphoid tissue (iBALT).Lack of Gαi2 leads to dilative cardiomyopathy and increased mortality in β1-adrenoceptor overexpressing miceGαi2 and Gαi3 Differentially Regulate Arrest from Flow and Chemotaxis in Mouse NeutrophilsBlunted apoptosis of erythrocytes in mice deficient in the heterotrimeric G-protein subunit Gαi2Gαi2 signaling is required for skeletal muscle growth, regeneration, and satellite cell proliferation and differentiation.Dibutyryl-cAMP attenuates pulmonary fibrosis by blocking myofibroblast differentiation via PKA/CREB/CBP signaling in rats with silicosis.Regulation of Chemokine Signal Integration by Activator of G-Protein Signaling 4 (AGS4).The impact of RGS and other G-protein regulatory proteins on Gαi-mediated signaling in immunity.Orai1 controls C5a-induced neutrophil recruitment in inflammation.Primary cilium migration depends on G-protein signalling control of subapical cytoskeleton.Competition for Gβγ dimers mediates a specific cross-talk between stimulatory and inhibitory G protein α subunits of the adenylyl cyclase in cardiomyocytes.Inhibition of Rho-associated kinase relieves C5a-induced proteinuria in murine nephrotic syndrome.Heterotrimeric G-protein subunit Gα contributes to agonist-sensitive apoptosis and degranulation in murine plateletsRXFP1 Receptor Activation by Relaxin-2 Induces Vascular Relaxation in Mice a Gα-Protein/PI3Kß/γ/Nitric Oxide-Coupled Pathway
P2860
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P2860
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@en
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@nl
type
label
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@en
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@nl
prefLabel
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@en
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@nl
P2093
P356
P1476
Gαi2 is the essential Gαi protein in immune complex-induced lung disease.
@en
P2093
Ana Novakovic
Bernd Nürnberg
Britta Gewecke
Franziska M Konrad
J Engelbert Gessner
Jörg Reutershan
Katja Pexa
Kristina Wiege
Reinhold E Schmidt
Roland P Piekorz
P304
P356
10.4049/JIMMUNOL.1201398
P407
P577
2012-12-05T00:00:00Z