Ultrafast action potentials mediate kilohertz signaling at a central synapse.
about
Strength and precision of neurotransmission at mammalian presynaptic terminalsON and OFF unipolar brush cells transform multisensory inputs to the auditory system.Implementation of linear sensory signaling via multiple coordinated mechanisms at central vestibular nerve synapses.Numbers of presynaptic Ca2+ channel clusters match those of functionally defined vesicular docking sites in single central synapses.Single granule cells excite Golgi cells and evoke feedback inhibition in the cochlear nucleus.Reduced endogenous Ca2+ buffering speeds active zone Ca2+ signalingKv3.1 uses a timely resurgent K(+) current to secure action potential repolarization.Excitatory Cerebellar Nucleocortical Circuit Provides Internal Amplification during Associative Conditioning.Synapse-Level Determination of Action Potential Duration by K(+) Channel Clustering in AxonsSynaptic plasticity in the auditory system: a review.Excitability tuning of axons in the central nervous system.The readily releasable pool of synaptic vesicles.Dendritic patch-clamp recordings from cerebellar granule cells demonstrate electrotonic compactness.Physical determinants of vesicle mobility and supply at a central synapseA use-dependent increase in release sites drives facilitation at calretinin-deficient cerebellar parallel-fiber synapses.Fast, Temperature-Sensitive and Clathrin-Independent Endocytosis at Central Synapses.Frequency-dependent mobilization of heterogeneous pools of synaptic vesicles shapes presynaptic plasticity.Synchrony, connectivity, and functional similarity in auditory midbrain local circuits.Sparse synaptic connectivity is required for decorrelation and pattern separation in feedforward networks.Kv3 Channels: Enablers of Rapid Firing, Neurotransmitter Release, and Neuronal Endurance.NMDA receptors amplify mossy fiber synaptic inputs at frequencies up to at least 750 Hz in cerebellar granule cells.Quantal Fluctuations in Central Mammalian Synapses: Functional Role of Vesicular Docking Sites.Complementary Tuning of Na+ and K+ Channel Gating Underlies Fast and Energy-Efficient Action Potentials in GABAergic Interneuron Axons.Kinetics of transmitter release at the calyx of Held synapse.Coupling the Structural and Functional Assembly of Synaptic Release SitesTwo-component latency distributions indicate two-step vesicular release at simple glutamatergic synapses
P2860
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P2860
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
@en
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
@nl
type
label
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
@en
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
@nl
prefLabel
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
@en
Ultrafast action potentials mediate kilohertz signaling at a central synapse.
@nl
P2093
P50
P1433
P1476
Ultrafast action potentials mediate kilohertz signaling at a central synapse
@en
P2093
Andreas Ritzau-Jost
Annika Rings
Johannes Hirrlinger
Ryuichi Shigemoto
P304
P356
10.1016/J.NEURON.2014.08.036
P407
P577
2014-09-11T00:00:00Z