Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
about
Unraveling the mechanism of BRCA2 in homologous recombinationRAD52 inactivation is synthetically lethal with deficiencies in BRCA1 and PALB2 in addition to BRCA2 through RAD51-mediated homologous recombinationMismatch repair during homologous and homeologous recombinationDual DNA-binding domains shape the interaction of Brh2 with DNA.Homologous recombination and its regulationDss1 release activates DNA binding potential in Brh2Brh2 domain function distinguished by differential cellular responses to DNA damage and replication stress.Dss1 Regulates Association of Brh2 with Rad51.Collaboration in the actions of Brh2 with resolving functions during DNA repair and replication stress in Ustilago maydis.
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Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
description
2010 nî lūn-bûn
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2010年の論文
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2010年論文
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2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
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2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
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2010年论文
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name
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@en
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@nl
type
label
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@en
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@nl
prefLabel
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@en
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@nl
P2093
P2860
P1476
Mutational analysis of Brh2 reveals requirements for compensating mediator functions.
@en
P2093
Milorad Kojic
Qingwen Zhou
William K Holloman
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P304
P356
10.1111/J.1365-2958.2010.07440.X
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P577
2010-11-02T00:00:00Z