The potassium channel KCa3.1 constitutes a pharmacological target for astrogliosis associated with ischemia stroke.

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The potassium channel KCa3.1 constitutes a pharmacological target for astrogliosis associated with ischemia stroke.

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

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2017年论文

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2017年论文

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The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

@en

The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

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label

The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

@en

The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

@nl

prefLabel

The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

@en

The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

@nl

P1476

The potassium channel KCa3.1 c ...... sociated with ischemia stroke.

@en

P2093

Gaoxian Chen

http://www.w3.org/2001/XMLSchema#string

Herbert M Geller

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Hongzhuan Chen

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Mengni Yi

http://www.w3.org/2001/XMLSchema#string

Qin Lu

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Tianjiao Wei

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Xiaoling Gao

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Yanxia Wang

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Zhihua Yu

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P2860

NIH Image to ImageJ: 25 years of image analysis

The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans

The Potassium Channel KCa3.1 Represents a Valid Pharmacological Target for Astrogliosis-Induced Neuronal Impairment in a Mouse Model of Alzheimer’s Disease

A cell-based microarrayed compound screening format for identifying agonists of G-protein-coupled receptors.

Neurotoxic reactive astrocytes are induced by activated microglia.

Inhibition of the K+ channel KCa3.1 ameliorates T cell-mediated colitis

Targeted inhibition of KCa3.1 attenuates TGF-β-induced reactive astrogliosis through the Smad2/3 signaling pathway.

Positive feedback regulation of agonist-stimulated endothelial Ca2+ dynamics by KCa3.1 channels in mouse mesenteric arteries.

The increased activity of TRPV4 channel in the astrocytes of the adult rat hippocampus after cerebral hypoxia/ischemia.

An aquaporin-4/transient receptor potential vanilloid 4 (AQP4/TRPV4) complex is essential for cell-volume control in astrocytes.

P2888

s12974-017-0973-8

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scholarly article

P356

10.1186/S12974-017-0973-8

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2017-10-16T00:00:00Z

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1092238669

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29037241

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