Dysfunctional nitric oxide signalling increases risk of myocardial infarction. - Wikidata - wikimedia - TriplyDB

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

http://www.wikidata.org/entity/Q42708482

about

Association claims in the sequencing era The impact of genome-wide association studies on the pathophysiology and therapy of cardiovascular disease Genetics of coronary artery disease and myocardial infarction Extending the translational potential of targeting NO/cGMP-regulated pathways in the CVS Genetic Architecture of Atherosclerosis in Mice: A Systems Genetics Analysis of Common Inbred Strains Prediction of Causal Candidate Genes in Coronary Artery Disease Loci Systems Medicine as an Emerging Tool for Cardiovascular Genetics Asymmetric dimethylarginine and cardiovascular risk: systematic review and meta-analysis of 22 prospective studies A comprehensive 1,000 Genomes-based genome-wide association meta-analysis of coronary artery disease Stroke Risk Factors, Genetics, and Prevention.Functional Characterization of the GUCY1A3 Coronary Artery Disease Risk Locus.Low-frequency and common genetic variation in ischemic stroke: The METASTROKE collaboration.Inherited dysfunctional nitric oxide signaling and the pathobiology of atherothrombotic disease.Hypertension reduces soluble guanylyl cyclase expression in the mouse aorta via the Notch signaling pathway Mutation in KERA identified by linkage analysis and targeted resequencing in a pedigree with premature atherosclerosis.Sensitivity to Sevoflurane anesthesia is decreased in mice with a congenital deletion of Guanylyl Cyclase-1 alpha.The fibrate gemfibrozil is a NO- and haem-independent activator of soluble guanylyl cyclase: in vitro studies.New Developments in Platelet Cyclic Nucleotide Signalling: Therapeutic Implications.Genetics of coronary artery disease: discovery, biology and clinical translation.Whole-exome sequencing in an extended family with myocardial infarction unmasks familial hypercholesterolemia.Inactivating mutations in NPC1L1 and protection from coronary heart disease Systematic cell-based phenotyping of missense alleles empowers rare variant association studies: a case for LDLR and myocardial infarction.Hemoglobin βCys93 is essential for cardiovascular function and integrated response to hypoxia.Cardiovascular and pharmacological implications of haem-deficient NO-unresponsive soluble guanylate cyclase knock-in mice.Hemoglobin induced NO/cGMP suppression Deteriorate Microcirculation via Pericyte Phenotype Transformation after Subarachnoid Hemorrhage in Rats A rare variant in MCF2L identified using exclusion linkage in a pedigree with premature atherosclerosis Whole-exome sequencing for the discovery of rare genetic variants that protect from coronary artery disease.The genetics of blood pressure regulation and its target organs from association studies in 342,415 individuals.Unbalanced Oxidant-Antioxidant Status: A Potential Therapeutic Target for Coronary Chronic Total Occlusion in Very Old Patients.Genetic testing in cardiovascular diseases.New therapies for coronary artery disease: genetics provides a blueprint.cGMP: transition from bench to bedside: a report of the 6th International Conference on cGMP Generators, Effectors and Therapeutic Implications.Next generation sequencing and the future of genetic diagnosis.Shared genetic aetiology of coronary artery disease and atherosclerotic stroke - 2015.Role of sGC-dependent NO signalling and myocardial infarction risk.Epidemiology in Germany-general development and personal experience.Molecular genetics of coronary artery disease.Glucose Homeostasis and Cardiovascular Alterations in Diabetes.NGS Technologies as a Turning Point in Rare Disease Research , Diagnosis and Treatment.Clinical relevance of cyclic GMP modulators: A translational success story of network pharmacology.

P2860

Q24273315-1E8023DA-4002-4F3A-B4FB-6CB55E0306CE Q26748869-31F6A5B4-EC98-4FB9-A103-EBE02ACC4974 Q26769994-6A56FD1D-D444-47AE-9798-3CEB88CF43FB Q27021649-3D920F53-8F86-43B2-92F2-44E86CE89315 Q27309881-0BC50C95-9D43-4DB7-8C1C-C283B8405D85 Q27342192-24240537-6E21-4910-9D52-93AC6C108C68 Q28069197-22EC47A0-1640-47CA-BD23-AD18854BC80E Q28087272-08DEBF0C-D9F7-4EEB-919B-EE917136B5C4 Q28267020-5A8E5D28-A4AB-4015-881C-5415880B7BF3 Q30238633-0F932C8E-8BF2-44CB-B3CC-9CDBD8CF5ECF Q30252419-41CBCA7F-0AB9-4BE2-B3A1-2C1D229BD685 Q30356365-9C652609-375D-40A1-8BCE-66E05637030B Q33642069-5935DEF7-D0B6-49FE-8F34-EB878DCE9726 Q33683635-03BE370B-74B6-404C-A2B0-E45A10CCC085 Q33690049-5135E104-BAF4-4E4C-A7F0-D4205F222E39 Q33801187-8861C8F9-41B4-48D1-9FB5-3BE4B0D4AB2D Q34042836-08792CFB-5114-4B79-8557-C842696A44C5 Q34532327-CC420AD4-ED60-4C00-A695-D088B2AA789C Q34553141-97C8D91D-481B-4872-AB5B-F65C8C813C7B Q34569742-361AE48E-DF89-4103-B98B-20FFB9F58DC7 Q35105445-56CD1DC0-2227-4C89-8550-89D3E827E9F3 Q35538234-F0E78A59-82E8-488D-9B12-C1BC44FD479A Q35644835-B234B0ED-0F8A-4AA6-A4BB-6585454BE1C8 Q36424922-46F8718D-68CE-4532-8D01-B2E9EDA70C91 Q36615945-9031C8A3-6249-4CE9-BB07-D2CB32D602DB Q36695830-37B117CB-4521-4334-B9B7-10E50A528B7D Q36832490-7C4D0D38-7140-4A77-9D97-6B4E9CCB1C1A Q37297746-B6961D5B-6EFB-4EC9-8B94-E9F8042631B6 Q37504067-B4F15FB6-F0F1-4696-ABF7-C62BB9FCDF52 Q38203394-AD49F077-938C-4744-9319-20ADCB5CCB84 Q38217732-477A1FF6-8A2B-48A1-A916-ACFFE81341C9 Q38220091-04356ADE-5E9D-4817-9570-4CE5B1E385CE Q38232771-7C0FB608-7726-4617-8C5F-84F14B625403 Q38359111-9846B765-0868-4C81-811F-697CA46FB654 Q38367012-861AD448-584B-4653-9545-86FB1DD33A03 Q38368774-37A06AC8-CA03-414F-9F32-1C5638B45537 Q38541814-1DB9F636-C3DA-4419-9272-492EBC4248C8 Q38596700-ABF3BFCC-3192-4693-90FD-1FECA489CF87 Q38655437-139D552E-F661-4462-BD0D-5B10A0EC3DA2 Q38696148-6515535E-0A64-4D6C-BDDB-B675F739157B

P2860

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

http://www.wikidata.org/entity/Q42708482

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en-gb

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@nl

type

label

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en-gb

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@nl

prefLabel

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en-gb

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@nl

P1433

Q42708482-90EC7D3A-9F92-48D6-9BD7-CA8D952F0CF8

P1476

Q42708482-4BF8C839-FEF2-42CB-AE44-B8E98603347A

P2093

Q42708482-0732E6F7-168F-49AC-B7B8-998A3F1F6B47

Q42708482-0D8CA3F6-2FD1-408B-A62B-07A437760222

Q42708482-0EB9CDC8-A995-4B65-A763-C7C8F61A5F80

Q42708482-14C0FBAB-82E9-4CFF-B4D5-33D64FE57CB0

Q42708482-1E4D1C7D-B419-48AA-88F4-72FEA906D798

Q42708482-23DC12CD-6B44-4942-B91E-08BF4D234E31

Q42708482-295E15B9-B8D4-47C0-8E02-59CC1CA7E1EA

Q42708482-33E6B219-0824-43E5-BC72-BC2E646D4E37

Q42708482-50B3DD15-BF8E-4252-B228-178753D420B5

Q42708482-679654A2-6CF5-448B-ADC0-416D95A88F31

P2860

Q42708482-061313E5-489C-494D-9346-461314519B4B

Q42708482-08ADE7E5-F239-4254-8E3D-93BE4AA1ED45

Q42708482-3CAA92B5-116C-42AB-A76A-FFDC191DB7F9

Q42708482-4CE2A711-5C0F-4332-9EE5-1690919F6154

Q42708482-58109679-9A4D-4F3B-AF9B-D4E454AC3115

Q42708482-59BE03DB-586A-483C-BE3B-F0557AA2B5D0

Q42708482-5FF416E6-D8C1-44BD-9831-F882C6E40D96

Q42708482-6C713DAC-502A-4675-8967-09CED8906340

Q42708482-7118BDA4-B92D-459E-8F9D-77E7AB508146

Q42708482-753C74E8-2235-4CB1-A516-3DA7E6027EFF

P2888

Q42708482-287162FD-8721-4215-9087-2CF01DD9A898

P304

Q42708482-3F2036D0-2ED7-41BC-8E73-B9749D74298B

P31

Q42708482-096D8933-B569-4C96-96E3-03097FAD28E3

P356

Q42708482-A4F561CC-AC97-43FD-9B5D-7CBD365943FD

P407

Q42708482-E9D13592-D3E8-407A-BBC6-673A7524C350

P433

Q42708482-00B463AE-ECEA-481C-B02B-82A17449CE86

P478

Q42708482-D7EA49B1-20C1-45DD-A55A-BD0AD3E5B452

P50

Q42708482-08BCC4EC-2673-4C12-92FC-874F70D49733

Q42708482-0AA9834C-8159-4147-A01E-6220AA7AFF2C

Q42708482-10DEDFB1-15D0-4C16-A288-920267568639

Q42708482-1403F064-4CD1-43E5-805F-B802A59D234D

Q42708482-7AA6DEB8-BBC2-4C57-93B7-AFCA0D124C23

Q42708482-90A149F9-161F-432B-A1E3-18A6D3496C49

Q42708482-9115A529-B1FC-49D2-A40F-E2740B3264FE

Q42708482-AAE75C71-E2F7-490E-B24C-64996F83C1D2

Q42708482-AB8490AC-8B6C-440C-BEA5-6AFE3BAE30EA

Q42708482-B74FC6A0-84E7-44AF-B4C6-7505E2959E93

P577

Q42708482-32220537-D2E4-4F83-BF84-1925C837CF43

P6179

Q42708482-D5C4D4EB-8AAE-4436-B223-02DC60738A01

P698

Q42708482-1287ECBA-6158-48AA-8396-A31DA57BA763

P921

Q42708482-C4AA3641-4452-48F3-A886-0E79C85F57F6

P356

P1433

P1476

Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

@en

P2093

Anja Medack

http://www.w3.org/2001/XMLSchema#string

Anthony J Balmforth

http://www.w3.org/2001/XMLSchema#string

CARDIoGRAM

http://www.w3.org/2001/XMLSchema#string

Christian Hengstenberg

http://www.w3.org/2001/XMLSchema#string

Christina Willenborg

http://www.w3.org/2001/XMLSchema#string

Christof Burgdorf

http://www.w3.org/2001/XMLSchema#string

Diana Braunholz

http://www.w3.org/2001/XMLSchema#string

Doris Koesling

http://www.w3.org/2001/XMLSchema#string

Elisabeth Graf

http://www.w3.org/2001/XMLSchema#string

Evanthia Mergia

http://www.w3.org/2001/XMLSchema#string

P2860

Human soluble guanylate cyclase: functional expression and revised isoenzyme family

A map of human genome variation from population-scale sequencing

The crystal structure of yeast CCT reveals intrinsic asymmetry of eukaryotic cytosolic chaperonins

The molecular architecture of the eukaryotic chaperonin TRiC/CCT

Concept of vulnerable/unstable plaque.

A method and server for predicting damaging missense mutations

Spare guanylyl cyclase NO receptors ensure high NO sensitivity in the vascular system

Genome-wide association study in Han Chinese identifies four new susceptibility loci for coronary artery disease.

Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease.

A new multipoint method for genome-wide association studies by imputation of genotypes

P2888

P304

432-436

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/NATURE12722

http://www.w3.org/2001/XMLSchema#string

P407

P433

7480

http://www.w3.org/2001/XMLSchema#string

P478

504

http://www.w3.org/2001/XMLSchema#string

P50

Elisabeth Steinhagen-Thiessen

Thomas Meitinger

Heribert Schunkert

Jeanette Erdmann

Sekar Kathiresan

Alistair Scott Hall

Markus M Nöthen

P577

2013-11-10T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1012295459

http://www.w3.org/2001/XMLSchema#string

P698

24213632

http://www.w3.org/2001/XMLSchema#string

P921