Wnt/β-catenin signaling is required to rescue midbrain dopaminergic progenitors and promote neurorepair in ageing mouse model of Parkinson's disease.
about
GSK-3β dysregulation contributes to parkinson's-like pathophysiology with associated region-specific phosphorylation and accumulation of tau and α-synucleinActivated astrocytes enhance the dopaminergic differentiation of stem cells and promote brain repair through bFGFEnhancing Beta-Catenin Activity via GSK3beta Inhibition Protects PC12 Cells against Rotenone Toxicity through Nurr1 Induction.Aging disrupts cell subpopulation dynamics and diminishes the function of mesenchymal stem cells.Blood transcriptomics of drug-naïve sporadic Parkinson's disease patients.Nurr1-Based Therapies for Parkinson's Disease.Functional Rescue of Dopaminergic Neuron Loss in Parkinson's Disease Mice After Transplantation of Hematopoietic Stem and Progenitor Cells.Pluripotent stem cells progressing to the clinic.Towards a Better Treatment Option for Parkinson's Disease: A Review of Adult Neurogenesis.Developing drugs that target the Wnt pathway: recent approaches in cancer and neurodegenerative diseases.Genetic Variants of Microtubule Actin Cross-linking Factor 1 (MACF1) Confer Risk for Parkinson's Disease.Dynamic Changes in the Nigrostriatal Pathway in the MPTP Mouse Model of Parkinson's DiseasePhysical Exercise Modulates L-DOPA-Regulated Molecular Pathways in the MPTP Mouse Model of Parkinson's Disease.Diversity of astroglial responses across human neurodegenerative disorders and brain aging.BDNF promotes human neural stem cell growth via GSK-3β-mediated crosstalk with the wnt/β-catenin signaling pathway.Nurr1 promotes intestinal regeneration after ischemia/reperfusion injury by inhibiting the expression of p21 (Waf1/Cip1).Microglia Polarization, Gene-Environment Interactions and Wnt/β-Catenin Signaling: Emerging Roles of Glia-Neuron and Glia-Stem/Neuroprogenitor Crosstalk for Dopaminergic Neurorestoration in Aged Parkinsonian Brain.Sporadic Parkinson's disease derived neuronal cells show disease-specific mRNA and small RNA signatures with abundant deregulation of piRNAs.Tau Pathology in Parkinson's DiseaseNeurodegenerative Diseases: Regenerative Mechanisms and Novel Therapeutic Approaches
P2860
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P2860
Wnt/β-catenin signaling is required to rescue midbrain dopaminergic progenitors and promote neurorepair in ageing mouse model of Parkinson's disease.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Wnt/β-catenin signaling is req ...... model of Parkinson's disease.
@en
Wnt/β-catenin signaling is req ...... model of Parkinson's disease.
@nl
type
label
Wnt/β-catenin signaling is req ...... model of Parkinson's disease.
@en
Wnt/β-catenin signaling is req ...... model of Parkinson's disease.
@nl
prefLabel
Wnt/β-catenin signaling is req ...... model of Parkinson's disease.
@en
Wnt/β-catenin signaling is req ...... model of Parkinson's disease.
@nl
P2860
P50
P356
P1433
P1476
Wnt/β-catenin signaling is req ...... e model of Parkinson's disease
@en
P2093
Stefano Pluchino
P2860
P304
P356
10.1002/STEM.1708
P577
2014-08-01T00:00:00Z