Persistent NF-kappaB activation in renal epithelial cells in a mouse model of HIV-associated nephropathy.
about
α-Actinin 4 potentiates nuclear factor κ-light-chain-enhancer of activated B-cell (NF-κB) activity in podocytes independent of its cytoplasmic actin binding functionThe ubiquitin-like protein FAT10 mediates NF-kappaB activationDown-regulation of NF-κB transcriptional activity in HIV-associated kidney disease by BRD4 inhibitionViral Subversion Mechanisms in Chronic Kidney Disease PathogenesisTM4SF10 and ADAP interaction in podocytes: role in Fyn activity and nephrin phosphorylationRegulation of Neph3 gene in podocytes--key roles of transcription factors NF-kappaB and Sp1.Renal HIV expression is unaffected by serum LPS levels in an HIV transgenic mouse model of LPS induced kidney injury.Biophysical properties of normal and diseased renal glomeruli.TNFR2 interposes the proliferative and NF-κB-mediated inflammatory response by podocytes to TNF-α.HIV-associated nephropathy: pathogenesis.Advances in our understanding of the pathogenesis of HIV-1 associated nephropathy in children.Tubular cell phenotype in HIV-associated nephropathy: role of phospholipid lysophosphatidic acid.Role of nicotinic and estrogen signaling during experimental acute and chronic bladder inflammation.Math6 expression during kidney development and altered expression in a mouse model of glomerulosclerosis.Suppression of HIV-1 replication by antiretroviral therapy improves renal function in persons with low CD4 cell counts and chronic kidney disease.Expression of HIV transgene aggravates kidney injury in diabetic miceNephrin deficiency activates NF-kappaB and promotes glomerular injury.HIV and the Kidney.Controversies in the pathogenesis of HIV-associated renal diseases.The molecular pathogenesis of HIV-1 associated nephropathy: recent advances.Functions of the podocyte proteins nephrin and Neph3 and the transcriptional regulation of their genes.Predictors of HIV-associated nephropathy.Accelerated development of collapsing glomerulopathy in mice congenic for the HIVAN1 locus.TGF-β1 induces podocyte injury through Smad3-ERK-NF-κB pathway and Fyn-dependent TRPC6 phosphorylation.
P2860
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P2860
Persistent NF-kappaB activation in renal epithelial cells in a mouse model of HIV-associated nephropathy.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
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2005年學術文章
@zh-hant
name
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@en
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@nl
type
label
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@en
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@nl
prefLabel
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@en
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@nl
P2860
P1476
Persistent NF-kappaB activatio ...... of HIV-associated nephropathy.
@en
P2093
Leslie A Bruggeman
Scott Martinka
P2860
P304
P356
10.1152/AJPRENAL.00208.2005
P577
2005-10-04T00:00:00Z