about
Anti-CD25 treatment depletes Treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis.Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection.In pulmonary paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology.Indoleamine 2,3-dioxygenase controls fungal loads and immunity in Paracoccidioidomicosis but is more important to susceptible than resistant hosts.Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps.Loss- and Gain-of-Function Approaches Indicate a Dual Role Exerted by Regulatory T Cells in Pulmonary Paracoccidioidomycosis.Tolerogenic Plasmacytoid Dendritic Cells Control Paracoccidioides brasiliensis Infection by Inducting Regulatory T Cells in an IDO-Dependent Manner.XTT Assay of Antifungal Activity.Toll-like receptors and fungal infections: the role of TLR2, TLR4 and MyD88 in paracoccidioidomycosis.Innate immunity to Paracoccidioides brasiliensis infection.Dectin-1 Activation Exacerbates Obesity and Insulin Resistance in the Absence of MyD88.Myeloid dendritic cells (DCs) of mice susceptible to paracoccidioidomycosis suppress T cell responses whereas myeloid and plasmacytoid DCs from resistant mice induce effector and regulatory T cells.Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosisLipoxin Inhibits Fungal Uptake by Macrophages and Reduces the Severity of Acute Pulmonary Infection Caused by Paracoccidioides brasiliensis.TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells.CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis.Toll-like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells.MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection.Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis.Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanismsTLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infectionAspergillus fumigatus calcium-responsive transcription factors regulate cell wall architecture promoting stress tolerance, virulence and caspofungin resistance
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Flávio V. Loures
@ast
Flávio V. Loures
@en
Flávio V. Loures
@es
Flávio V. Loures
@nl
Flávio V. Loures
@sl
type
label
Flávio V. Loures
@ast
Flávio V. Loures
@en
Flávio V. Loures
@es
Flávio V. Loures
@nl
Flávio V. Loures
@sl
prefLabel
Flávio V. Loures
@ast
Flávio V. Loures
@en
Flávio V. Loures
@es
Flávio V. Loures
@nl
Flávio V. Loures
@sl
P106
P1153
24080690100
P21
P31
P496
0000-0001-7711-4063