Retinoblastoma protein (pRb), but not p107 or p130, is required for maintenance of enterocyte quiescence and differentiation in small intestine.
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MicroRNA mir-16 is anti-proliferative in enterocytes and exhibits diurnal rhythmicity in intestinal cryptsThe Pathogenesis of Resection-Associated Intestinal AdaptationTransgenic Soybean Production of Bioactive Human Epidermal Growth Factor (EGF)Omcg1 is critically required for mitosis in rapidly dividing mouse intestinal progenitors and embryonic stem cells.Up-regulation of hypoxia-inducible factor 1 alpha and hemodynamic responses following massive small bowel resection.PIAS1 is a GATA4 SUMO ligase that regulates GATA4-dependent intestinal promoters independent of SUMO ligase activity and GATA4 sumoylation.GATA4 Is Sufficient to Establish Jejunal Versus Ileal Identity in the Small Intestine.Disruption of retinoblastoma protein expression in the intestinal epithelium impairs lipid absorptionInsulin-like growth factor 2 and its enterocyte receptor are not required for adaptation in response to massive small bowel resection.Coordinate down-regulation of adenylyl cyclase isoforms and the stimulatory G protein (G(s)) in intestinal epithelial cell differentiation.The role of enteral fat as a modulator of body composition after small bowel resectionEpithelial cell specific Raptor is required for initiation of type 2 mucosal immunity in small intestineDissecting the unique role of the retinoblastoma tumor suppressor during cellular senescenceGATA4 and GATA6 regulate intestinal epithelial cytodifferentiation during development.IGF-2 is necessary for retinoblastoma-mediated enhanced adaptation after small-bowel resectionGeneration of a Retinoblastoma (Rb)1-inducible dominant-negative (DN) mouse model.The rb pathway and cancer therapeuticsHigh-fat diet enhances villus growth during the adaptation response to massive proximal small bowel resection.The effect of impaired angiogenesis on intestinal function following massive small bowel resection.Both epidermal growth factor and insulin-like growth factor receptors are dispensable for structural intestinal adaptation.p38 MAPK regulates Bax activity and apoptosis in enterocytes at baseline and after intestinal resection.Deletion of p38-alpha mitogen-activated protein kinase within the intestinal epithelium promotes colon tumorigenesis.Enterocyte expression of epidermal growth factor receptor is not required for intestinal adaptation in response to massive small bowel resectionRibosome biogenesis dysfunction leads to p53-mediated apoptosis and goblet cell differentiation of mouse intestinal stem/progenitor cellsFasting protects mice from lethal DNA damage by promoting small intestinal epithelial stem cell survival.RB1, development, and cancer.High-protein diet improves postoperative weight gain after massive small-bowel resection.Pocket proteins critically regulate cell cycle exit of the trabecular myocardium and the ventricular conduction systemRegulation of retinoblastoma protein (Rb) by p21 is critical for adaptation to massive small bowel resection.Simian virus 40 T-antigen-mediated gene regulation in enterocytes is controlled primarily by the Rb-E2F pathwayInhibition of CDK4/6 protects against radiation-induced intestinal injury in mice.Cdc42 coordinates proliferation, polarity, migration, and differentiation of small intestinal epithelial cells in mice.IGF-2 mediates intestinal mucosal hyperplasia in retinoblastoma protein (Rb)-deficient mice.Intestinal Epithelial-Specific mTORC1 Activation Enhances Intestinal Adaptation After Small Bowel Resection.RIP140 increases APC expression and controls intestinal homeostasis and tumorigenesis.Liver steatosis induced by small bowel resection is prevented by oral vancomycin.Adaptation: paradigm for the gut and an academic career.Viral oncogene expression in the stem/progenitor cell compartment of the mouse intestine induces adenomatous polyps.Retinoblastoma family proteins have distinct functions in pulmonary epithelial cells in vivo critical for suppressing cell growth and tumorigenesisAn In Vitro Model of Cellular Quiescence in Primary Human Dermal Fibroblasts.
P2860
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P2860
Retinoblastoma protein (pRb), but not p107 or p130, is required for maintenance of enterocyte quiescence and differentiation in small intestine.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@en
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@nl
type
label
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@en
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@nl
prefLabel
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@en
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@nl
P2093
P2860
P356
P1476
Retinoblastoma protein (pRb), ...... rentiation in small intestine.
@en
P2093
Brad W Warner
Rajalakshmi Nair
Shannon Longshore
P2860
P304
P356
10.1074/JBC.M806133200
P407
P577
2008-11-03T00:00:00Z