Concerted activities of distinct H4K20 methyltransferases at DNA double-strand breaks regulate 53BP1 nucleation and NHEJ-directed repair.
about
Writers, Readers, and Erasers of Histone Ubiquitylation in DNA Double-Strand Break RepairDNA double-strand break repair: a tale of pathway choices.Direct involvement of retinoblastoma family proteins in DNA repair by non-homologous end-joining.Loss of the N-terminal methyltransferase NRMT1 increases sensitivity to DNA damage and promotes mammary oncogenesisDNA double strand break repair pathway choice: a chromatin based decision?Histone H4 Lysine 20 (H4K20) Methylation, Expanding the Signaling Potential of the Proteome One Methyl Moiety at a Time.A PALB2-interacting domain in RNF168 couples homologous recombination to DNA break-induced chromatin ubiquitylation.Targeting chromatin to improve radiation response.The SUV4-20 inhibitor A-196 verifies a role for epigenetics in genomic integrity.Biological function and regulation of histone and non-histone lysine methylation in response to DNA damage.SET8 methyltransferase activity during the DNA double-strand break response is required for recruitment of 53BP1.Epigenetic modifications in multiple myeloma: recent advances on the role of DNA and histone methylation.Beyond histones - the expanding roles of protein lysine methylation.Epigenetic regulatory mutations and epigenetic therapy for multiple myeloma.Kmt5a Controls Hepatic Metabolic Pathways by Facilitating RNA Pol II Release from Promoter-Proximal Regions.G9a coordinates with the RPA complex to promote DNA damage repair and cell survival.Class I histone deacetylase inhibitors inhibit the retention of BRCA1 and 53BP1 at the site of DNA damage.Epigenetic therapy with inhibitors of histone methylation suppresses DNA damage signaling and increases glioma cell radiosensitivityHistone peptide microarray screen of chromo and Tudor domains defines new histone lysine methylation interactions.PR-Set7 deficiency limits uterine epithelial population growth hampering postnatal gland formation in mice.DICER- and MMSET-catalyzed H4K20me2 recruits the nucleotide excision repair factor XPA to DNA damage sites.Reading chromatin signatures after DNA double-strand breaks.MMSET/WHSC1 enhances DNA damage repair leading to an increase in resistance to chemotherapeutic agents.Replication-Coupled Dilution of H4K20me2 Guides 53BP1 to Pre-replicative Chromatin.The HTLV-1 basic leucine zipper factor (HBZ) attenuates repair of double-stranded DNA breaks via non-homologous end joining (NHEJ).Histone methylation in DNA repair and clinical practice: new findings during the past 5-years.TGF-β signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac aging.
P2860
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P2860
Concerted activities of distinct H4K20 methyltransferases at DNA double-strand breaks regulate 53BP1 nucleation and NHEJ-directed repair.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Concerted activities of distin ...... tion and NHEJ-directed repair.
@en
Concerted activities of distin ...... tion and NHEJ-directed repair.
@nl
type
label
Concerted activities of distin ...... tion and NHEJ-directed repair.
@en
Concerted activities of distin ...... tion and NHEJ-directed repair.
@nl
prefLabel
Concerted activities of distin ...... tion and NHEJ-directed repair.
@en
Concerted activities of distin ...... tion and NHEJ-directed repair.
@nl
P2093
P2860
P1433
P1476
Concerted activities of distin ...... tion and NHEJ-directed repair.
@en
P2093
Judd C Rice
Kyoko Yokomori
Lauren M Congdon
Tanya Spektor
Xiaodong Kong
P2860
P304
P356
10.1016/J.CELREP.2014.06.013
P577
2014-07-04T00:00:00Z