Variations in Jun and Fos protein expression and AP-1 activity in cycling, resting and stimulated fibroblasts.
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Identification of SWI.SNF complex subunit BAF60a as a determinant of the transactivation potential of Fos/Jun dimersRegulation of two JunD isoforms by Jun N-terminal kinasesCharacterization of the MN/CA 9 promoter proximal region: a role for specificity protein (SP) and activator protein 1 (AP1) factorsInduction of c-jun and TGF-beta 1 in Fischer 344 rats during amiodarone-induced pulmonary fibrosisFra-1 promotes growth and survival in RAS-transformed thyroid cells by controlling cyclin A transcriptionThe small GTP-binding protein Cdc42 is required for nerve growth factor withdrawal-induced neuronal deathRegulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion.In vitro selection of Jun-associated proteins using mRNA display.Deoxycholate induces COX-2 expression via Erk1/2-, p38-MAPK and AP-1-dependent mechanisms in esophageal cancer cells.Stress-activated protein kinases are negatively regulated by cell density.The transcription factor Ap-1 regulates monkey 20α-hydroxysteroid dehydrogenase promoter activity in CHO cells.An extracellular signal-regulated kinase 1- and 2-dependent program of chromatin trafficking of c-Fos and Fra-1 is required for cyclin D1 expression during cell cycle reentry.The mammalian Jun proteins: redundancy and specificity.Promoter specificity and biological activity of tethered AP-1 dimers.Tethering of the conserved piggyBac transposase fusion protein CSB-PGBD3 to chromosomal AP-1 proteins regulates expression of nearby genes in humans.Cell cycle-dependent variations in c-Jun and JunB phosphorylation: a role in the control of cyclin D1 expression.JunD and JunB integrate prostaglandin E2 activation of breast cancer-associated proximal aromatase promoters.Study on the apoptosis mechanism induced by T-2 toxin.Signal transduction mediated by the Ras/Raf/MEK/ERK pathway from cytokine receptors to transcription factors: potential targeting for therapeutic intervention.JunD protects against chronic kidney disease by regulating paracrine mitogens.JunB breakdown in mid-/late G2 is required for down-regulation of cyclin A2 levels and proper mitosisThe oncogenic transcription factor c-Jun regulates glutaminase expression and sensitizes cells to glutaminase-targeted therapy.Cooperation of protein kinase A and Ras/ERK signaling pathways is required for AP-1-mediated activation of fibroblast growth factor-inducible response element (FiRE).Basic fibroblast growth factor decreases elastin gene transcription through an AP1/cAMP-response element hybrid site in the distal promoter.C3 toxin activates the stress signaling pathways, JNK and p38, but antagonizes the activation of AP-1 in rat-1 cells.Impaired ionizing radiation-induced activation of a nuclear signal essential for phosphorylation of c-Jun by dually phosphorylated c-Jun amino-terminal kinases in ataxia telangiectasia fibroblasts.Signal transduction pathways and modulation of gene activity.A p38 Mitogen-Activated Protein Kinase-Regulated Myocyte Enhancer Factor 2-β-Catenin Interaction Enhances Canonical Wnt Signaling.GSK3-SCF(FBXW7) targets JunB for degradation in G2 to preserve chromatid cohesion before anaphase.The repertoire of fos and jun proteins expressed during the G1 phase of the cell cycle is determined by the duration of mitogen-activated protein kinase activation.Modulation of oxidative stress responses in the human retinal pigment epithelium following treatment with vitamin C.Ubiquitin-independent proteasomal degradation of Fra-1 is antagonized by Erk1/2 pathway-mediated phosphorylation of a unique C-terminal destabilizer.Accelerated cell cycle progression in osteoblasts overexpressing the c-fos proto-oncogene: induction of cyclin A and enhanced CDK2 activity.c-Fos activated phospholipid synthesis is required for neurite elongation in differentiating PC12 cells.Identification and characterization of JunD missense mutants that lack menin binding.Control of the adrenocortical cell cycle: interaction between FGF2 and ACTH.Three activator protein-1-binding sites bound by the Fra-2.JunD complex cooperate for the regulation of murine laminin alpha3A (lama3A) promoter activity by transforming growth factor-beta.Cross-species characterization of the promoter region of the cystic fibrosis transmembrane conductance regulator gene reveals multiple levels of regulation.An enhanceosome containing the Jun B/Fra-2 heterodimer and the HMG-I(Y) architectural protein controls HPV 18 transcription.Attenuated expression of the serum responsive T1 gene in ras transformed fibroblasts due to the inhibition of c-fos gene activity.
P2860
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P2860
Variations in Jun and Fos protein expression and AP-1 activity in cycling, resting and stimulated fibroblasts.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@en
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@nl
type
label
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@en
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@nl
prefLabel
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@en
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@nl
P2093
P356
P1433
P1476
Variations in Jun and Fos prot ...... ng and stimulated fibroblasts.
@en
P2093
P2888
P304
P356
10.1038/SJ.ONC.1200901
P407
P577
1997-02-01T00:00:00Z
P5875
P6179
1009404252