Mice can recover from pulmonary influenza virus infection in the absence of class I-restricted cytotoxic T cells.
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P1343
Patterns of predicted T-cell epitopes associated with antigenic drift in influenza H3N2 hemagglutininA pandemic influenza H1N1 live vaccine based on modified vaccinia Ankara is highly immunogenic and protects mice in active and passive immunizationsCD8(+) T cell-mediated injury in vivo progresses in the absence of effector T cells.Receptor for advanced glycation end products is detrimental during influenza A virus pneumonia.A whole virus pandemic influenza H1N1 vaccine is highly immunogenic and protective in active immunization and passive protection mouse models.Memory CD4 T cells direct protective responses to influenza virus in the lungs through helper-independent mechanisms.Hallmarks of CD4 T cell immunity against influenzaDNA vaccination elicits protective immune responses against pandemic and classic swine influenza viruses in pigs.Protective antiviral antibody responses in a mouse model of influenza virus infection require TACIResistance to and recovery from lethal influenza virus infection in B lymphocyte-deficient mice.Structural and functional consequences of alveolar cell recognition by CD8(+) T lymphocytes in experimental lung disease.Influenza virus-specific CD4+ T helper type 2 T lymphocytes do not promote recovery from experimental virus infection.Consequences of immunodominant epitope deletion for minor influenza virus-specific CD8+-T-cell responses.Role of interleukin-12 in primary influenza virus infectionProtective CD4+ and CD8+ T cells against influenza virus induced by vaccination with nucleoprotein DNAImmunization of pigs with a particle-mediated DNA vaccine to influenza A virus protects against challenge with homologous virusVaccination with M2e-based multiple antigenic peptides: characterization of the B cell response and protection efficacy in inbred and outbred mice.Ribavirin and IFN-alpha combination therapy induces CD4+ T-cell proliferation and Th1 cytokine secretion in patients with chronic hepatitis BCoinfection with Streptococcus pneumoniae modulates the B cell response to influenza virusEnvironmental and antigen receptor-derived signals support sustained surveillance of the lungs by pathogen-specific cytotoxic T lymphocytes.Protection and compensation in the influenza virus-specific CD8+ T cell responseVirus-neutralizing antibodies of immunoglobulin G (IgG) but not of IgM or IgA isotypes can cure influenza virus pneumonia in SCID mice.The B-cell response in lymphoid tissue of mice immunized with various antigenic forms of the influenza virus hemagglutinin.Antibody-forming cell response to virus challenge in mice immunized with DNA encoding the influenza virus hemagglutininImmune CD4+ T cells promote the clearance of influenza virus from major histocompatibility complex class II -/- respiratory epitheliumCellular mechanisms involved in protection and recovery from influenza virus infection in immunodeficient mice.Oral immunization with a replication-deficient recombinant vaccinia virus protects mice against influenzaA pulmonary influenza virus infection in SCID mice can be cured by treatment with hemagglutinin-specific antibodies that display very low virus-neutralizing activity in vitro.Transgenic mice lacking class I major histocompatibility complex-restricted T cells have delayed viral clearance and increased mortality after influenza virus challenge.Human monoclonal antibodies against a plethora of viral pathogens from single combinatorial libraries.CD4(+) T cell subsets during virus infection. Protective capacity depends on effector cytokine secretion and on migratory capability.B-1 and B-2 cell-derived immunoglobulin M antibodies are nonredundant components of the protective response to influenza virus infectionOvalbumin sensitization changes the inflammatory response to subsequent parainfluenza infection. Eosinophils mediate airway hyperresponsiveness, m(2) muscarinic receptor dysfunction, and antiviral effectsNeutrophils play an essential role in cooperation with antibody in both protection against and recovery from pulmonary infection with influenza virus in mice.Enhanced establishment of a virus carrier state in adult CD4+ T-cell-deficient mice.Vesicular stomatitis virus Indiana glycoprotein as a T-cell-dependent and -independent antigen.Novel features of the respiratory tract T-cell response to influenza virus infection: lung T cells increase expression of gamma interferon mRNA in vivo and maintain high levels of mRNA expression for interleukin-5 (IL-5) and IL-10.Divergence between cytotoxic effector function and tumor necrosis factor alpha production for inflammatory CD4+ T cells from mice with Sendai virus pneumonia.Splenic priming of virus-specific CD8 T cells following influenza virus infectionDendritic cells and B cells maximize mucosal Th1 memory response to herpes simplex virus
P2860
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P2860
Mice can recover from pulmonary influenza virus infection in the absence of class I-restricted cytotoxic T cells.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Mice can recover from pulmonar ...... -restricted cytotoxic T cells.
@en
Mice can recover from pulmonar ...... -restricted cytotoxic T cells.
@nl
type
label
Mice can recover from pulmonar ...... -restricted cytotoxic T cells.
@en
Mice can recover from pulmonar ...... -restricted cytotoxic T cells.
@nl
prefLabel
Mice can recover from pulmonar ...... -restricted cytotoxic T cells.
@en
Mice can recover from pulmonar ...... -restricted cytotoxic T cells.
@nl
P2093
P1476
Mice can recover from pulmonar ...... I-restricted cytotoxic T cells
@en
P2093
Palladino G
Scherle PA
P304
P407
P4510
P577
1992-01-01T00:00:00Z