Expression of APO-1 (CD95), a member of the NGF/TNF receptor superfamily, in normal and neoplastic colon epithelium.
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Carbon nanotubes induce apoptosis resistance of human lung epithelial cells through FLICE-inhibitory proteinCarcinoembryonic antigen family members CEACAM6 and CEACAM7 are differentially expressed in normal tissues and oppositely deregulated in hyperplastic colorectal polyps and early adenomasRegulation of the metastatic cell phenotype by sialylated glycansDecitabine and vorinostat cooperate to sensitize colon carcinoma cells to Fas ligand-induced apoptosis in vitro and tumor suppression in vivoMatrilysin [MMP-7] expression selects for cells with reduced sensitivity to apoptosisCeramide mediates FasL-induced caspase 8 activation in colon carcinoma cells to enhance FasL-induced cytotoxicity by tumor-specific cytotoxic T lymphocytesFAS/FASL expression profile as a prognostic marker in squamous cell carcinoma of the oral cavity.Expression and Y435-phosphorylation of Abelson interactor 1 (Abi1) promotes tumour cell adhesion, extracellular matrix degradation and invasion by colorectal carcinoma cells.Cell surface molecules and their prognostic values in assessing colorectal carcinomas.Tumour necrosis factor superfamily members in the pathogenesis of inflammatory bowel diseaseOncogenic Ras inhibits Fas ligand-mediated apoptosis by downregulating the expression of Fas.Apoptosis: implications for inflammatory bowel disease.Isolation and characterization of intestinal epithelial cells from normal and SIV-infected rhesus macaquesReview article: a conceptual approach to understanding the molecular mechanisms of cancer development in Barrett's oesophagus.Expression of Abelson interactor 1 (Abi1) correlates with inflammation, KRAS mutation and adenomatous change during colonic carcinogenesis.Growth of human colorectal cancer SW1116 cells is inhibited by cytokine-induced killer cells.Alteration of DNA methylation in gastrointestinal carcinogenesis.Sialylation of the Fas death receptor by ST6Gal-I provides protection against Fas-mediated apoptosis in colon carcinoma cellsFas ligand upregulation is an early event in colonic carcinogenesis.MicroRNA 196B regulates FAS-mediated apoptosis in colorectal cancer cellsImpaired CD95 expression predisposes for recurrence in curatively resected colon carcinoma: clinical evidence for immunoselection and CD95L mediated control of minimal residual disease.Expression of CD95 antigen and Bcl-2 protein in non-Hodgkin's lymphomas and Hodgkin's disease.Colon cancer: genomics and apoptotic events.Corruption of the Fas pathway delays the pulmonary clearance of murine osteosarcoma cells, enhances their metastatic potential, and reduces the effect of aerosol gemcitabine.H3K9 Trimethylation Silences Fas Expression To Confer Colon Carcinoma Immune Escape and 5-Fluorouracil Chemoresistance.Increased expression but not sensitivity to Fas/CD95 in glioblastoma cells depleted of mitochondrial DNA.Targeting the Fas/Fas ligand pathway in cancer.Sustained polymorphonuclear leukocyte transmigration induces apoptosis in T84 intestinal epithelial cellsThymineless death in colon carcinoma cells is mediated via fas signaling.Dual role for Fas ligand in the initiation of and recovery from experimental allergic encephalomyelitis.c-FLIP mediates resistance of Hodgkin/Reed-Sternberg cells to death receptor-induced apoptosis.Fas and Fas ligand interactions suppress melanoma lung metastasis.The Fas counterattack: a molecular mechanism of tumor immune privilege.Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL-induced apoptosis in vitro and in vivo.Quantitation of Fas and Fas ligand gene expression in human ovarian, cervical and endometrial carcinomas using real-time quantitative RT-PCR.Endothelin-receptor antagonists are proapoptotic and antiproliferative in human colon cancer cells.The clinical significance of autoantibodies in gastrointestinal malignancies: an overview.Multifaceted targeting in cancer: the recent cell death players meet the usual oncogene suspects.VEGF165 is necessary to the metastatic potential of Fas(-) osteosarcoma cells but will not rescue the Fas(+) cellsColonic involvement in Stevens-Johnson syndrome.
P2860
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P1343
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P2860
Expression of APO-1 (CD95), a member of the NGF/TNF receptor superfamily, in normal and neoplastic colon epithelium.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@en
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@nl
type
label
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@en
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@nl
prefLabel
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@en
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@nl
P2093
P4510
P356
P1476
Expression of APO-1 (CD95), a ...... d neoplastic colon epithelium.
@en
P2093
A Quentmeier
F Leithäuser
P H Krammer
S Brüderlein
P304
P356
10.1002/IJC.2910570314
P577
1994-05-01T00:00:00Z