Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
about
TPM3-ALK and TPM4-ALK oncogenes in inflammatory myofibroblastic tumorsCalponin repeats regulate actin filament stability and formation of podosomes in smooth muscle cells.Post-transcriptional down-regulation of ROCKI/Rho-kinase through an MEK-dependent pathway leads to cytoskeleton disruption in Ras-transformed fibroblasts.Opposing roles of the extracellular signal-regulated kinase and p38 mitogen-activated protein kinase cascades in Ras-mediated downregulation of tropomyosin.Tropomyosin-based regulation of the actin cytoskeleton in time and space.Overexpression of kinase suppressor of Ras upregulates the high-molecular-weight tropomyosin isoforms in ras-transformed NIH 3T3 fibroblastsInterior decoration: tropomyosin in actin dynamics and cell migration.Tropomyosin isoforms and reagentsHypoxia alters the recruitment of tropomyosins into the actin stress fibres of neuroblastoma cells.Expression of tropomyosin 2 gene isoforms in human breast cancer cell lines.Functional analysis of the actin-binding protein, tropomyosin 1, in neuroblastoma.From the Cover: Neutralization of terminal differentiation in gliomagenesis.New insights into the regulation of the actin cytoskeleton by tropomyosin.Tropomyosin Structure, Function, and Interactions: A Dynamic Regulator.The Ras-ERK pathway modulates cytoskeleton organization, cell motility and lung metastasis signature genes in MDA-MB-231 LM2.Downregulation of tropomyosin-1 in squamous cell carcinoma of esophagus, the role of Ras signaling and methylation.Increased expression of dihydrodiol dehydrogenase induces resistance to cisplatin in human ovarian carcinoma cells.Repression of in vivo growth of Myc/Ras transformed tumor cells by Mad1.Cytoskeletal organization in tropomyosin-mediated reversion of ras-transformation: Evidence for Rho kinase pathway.Myocardin-related transcription factor A (MRTF-A) activity-dependent cell adhesion is correlated to focal adhesion kinase (FAK) activity.N terminus is essential for tropomyosin functions: N-terminal modification disrupts stress fiber organization and abolishes anti-oncogenic effects of tropomyosin-1.Carcinoembryonic antigen cell adhesion molecule 1 directly associates with cytoskeleton proteins actin and tropomyosin.
P2860
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P2860
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@en
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@nl
type
label
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@en
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@nl
prefLabel
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@en
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@nl
P2093
P2860
P356
P1433
P1476
Suppression of src-induced transformed phenotype by expression of tropomyosin-1.
@en
P2093
Harindranath N
Masuelli L
P2860
P2888
P304
P356
10.1038/SJ.ONC.1202264
P407
P577
1999-03-01T00:00:00Z
P5875
P6179
1031525685