A natural hepatocyte growth factor/scatter factor autocrine loop in myoblast cells and the effect of the constitutive Met kinase activation on myogenic differentiation.
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Early-age heat exposure affects skeletal muscle satellite cell proliferation and differentiation in chicksPAX3-FOXO1 fusion gene in rhabdomyosarcomaSatellite cells in human skeletal muscle plasticityc-MET regulates myoblast motility and myocyte fusion during adult skeletal muscle regenerationLamin A/C and emerin are critical for skeletal muscle satellite cell differentiationUnderstanding the Interplay between Expression, Mutation and Activity of ALK Receptor in Rhabdomyosarcoma Cells for Clinical Application of Small-Molecule InhibitorsStable Ectopic Expression of ST6GALNAC5 Induces Autocrine MET Activation and Anchorage-Independence in MDCK CellsA novel mechanism of sequestering fibroblast growth factor 2 by glypican in lipid rafts, allowing skeletal muscle differentiationThe methyltransferase SMYD3 mediates the recruitment of transcriptional cofactors at the myostatin and c-Met genes and regulates skeletal muscle atrophyThe decreased metastatic potential of rhabdomyosarcoma cells obtained through MET receptor downregulation and the induction of differentiationMinimally invasive approach to the repair of injured skeletal muscle with a shape-memory scaffold.Antisense inhibition of decorin expression in myoblasts decreases cell responsiveness to transforming growth factor beta and accelerates skeletal muscle differentiation.Syndecan-1 expression inhibits myoblast differentiation through a basic fibroblast growth factor-dependent mechanism.Magic-factor 1, a partial agonist of Met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis.Inhibition of extracellular matrix assembly induces the expression of osteogenic markers in skeletal muscle cells by a BMP-2 independent mechanism.Real-time quantitative measurement of autocrine ligand binding indicates that autocrine loops are spatially localized.High concentrations of HGF inhibit skeletal muscle satellite cell proliferation in vitro by inducing expression of myostatin: a possible mechanism for reestablishing satellite cell quiescence in vivo.Satellite cells and the muscle stem cell nicheNeuregulin stimulates myogenic differentiation in an autocrine manner.Oncogenic mutants of RON and MET receptor tyrosine kinases cause activation of the beta-catenin pathway.In vitro indeterminate teleost myogenesis appears to be dependent on Pax3Non-agonistic bivalent antibodies that promote c-MET degradation and inhibit tumor growth and others specific for tumor related c-MET.Constitutive activation of MET signaling impairs myogenic differentiation of rhabdomyosarcoma and promotes its development and progression.Conditional activation of MET in differentiated skeletal muscle induces atrophy.Insulin-like growth factors, hepatocyte growth factor and transforming growth factor-alpha in mouse tongue myogenesis.A novel isoform of met receptor tyrosine kinase blocks hepatocyte growth factor/Met signaling and stimulates skeletal muscle cell differentiation.The hepatocyte growth factor/c-Met signaling pathway as a therapeutic target to inhibit angiogenesis.Localization of Magic-F1 transgene, involved in muscular hypertrophy, during early myogenesisFGFR4 and its novel splice form in myogenic cells: Interplay of glycosylation and tyrosine phosphorylation.Sphingosine kinase/sphingosine 1-phosphate axis: a new player for insulin-like growth factor-1-induced myoblast differentiation.Interferon regulatory factor-2 is a transcriptional activator in muscle where It regulates expression of vascular cell adhesion molecule-1.Mesenchymal stem cells and myoblast differentiation under HGF and IGF-1 stimulation for 3D skeletal muscle tissue engineering.MicroRNA reexpression as differentiation therapy in cancerThe muscle-specific microRNA miR-206 blocks human rhabdomyosarcoma growth in xenotransplanted mice by promoting myogenic differentiationMolecular and cellular biology of rhabdomyosarcoma.Receptor tyrosine kinases as therapeutic targets in rhabdomyosarcoma.Dose-dependent modulation of myogenesis by HGF: implications for c-Met expression and downstream signalling pathways.α-Syntrophin is required for the hepatocyte growth factor-induced migration of cultured myoblasts.Cell delivery of Met docking site peptides inhibit angiogenesis and vascular tumor growth.The MET receptor tyrosine kinase contributes to invasive tumour growth in rhabdomyosarcomas.
P2860
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P2860
A natural hepatocyte growth factor/scatter factor autocrine loop in myoblast cells and the effect of the constitutive Met kinase activation on myogenic differentiation.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@en
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@nl
type
label
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@en
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@nl
prefLabel
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@en
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@nl
P2093
P2860
P356
P1476
A natural hepatocyte growth fa ...... n on myogenic differentiation.
@en
P2093
P2860
P304
P356
10.1083/JCB.137.5.1057
P407
P577
1997-06-01T00:00:00Z