Hutchinson-Gilford progeria syndrome alters nuclear shape and reduces cell motility in three dimensional model substrates.
about
Physical biology in cancer. 5. The rocky road of metastasis: the role of cytoskeletal mechanics in cell migratory response to 3D matrix topographyNuclear deformability constitutes a rate-limiting step during cell migration in 3-D environmentsFrom repair to regeneration: biomaterials to reprogram the meniscus wound microenvironment.Nuclear mechanics in cancerOn-chip assay of the effect of topographical microenvironment on cell growth and cell-cell interactions during wound healing.Nesprin-2G, a Component of the Nuclear LINC Complex, Is Subject to Myosin-Dependent Tension.Squish and squeeze-the nucleus as a physical barrier during migration in confined environments.Vertical nanopillars for in situ probing of nuclear mechanics in adherent cells.Nuclear mechanics and mechanotransduction in health and disease.Prelamin A Accumulation Attenuates Rac1 Activity and Increases the Intrinsic Migrational Persistence of Aged Vascular Smooth Muscle Cells.Broken nuclei--lamins, nuclear mechanics, and disease.Cancer cell motility: lessons from migration in confined spaces.Physical influences of the extracellular environment on cell migration.Lamins at the crossroads of mechanosignalingAn insight into morphometric descriptors of cell shape that pertain to regenerative medicine.Hutchinson-Gilford progeria syndrome as a model for vascular aging.Lmna knockout mouse embryonic fibroblasts are less contractile than their wild-type counterparts.Recent advances in understanding nuclear size and shape.Enhanced SRSF5 Protein Expression Reinforces Lamin A mRNA Production in HeLa Cells and Fibroblasts of Progeria Patients.A Tissue Engineered Blood Vessel Model of Hutchinson-Gilford Progeria Syndrome Using Human iPSC-derived Smooth Muscle CellsNuclear stiffening inhibits migration of invasive melanoma cells.Optimization of the Wound Scratch Assay to Detect Changes in Murine Mesenchymal Stromal Cell Migration After Damage by Soluble Cigarette Smoke Extract.Age of heart disease presentation and dysmorphic nuclei in patients with LMNA mutations.Role of the Nucleus as a Sensor of Cell Environment Topography.Nuclear stiffening and chromatin softening with progerin expression leads to an attenuated nuclear response to force.Maturation State and Matrix Microstructure Regulate Interstitial Cell Migration in Dense Connective Tissues.Early Passage Dependence of Mesenchymal Stem Cell Mechanics Influences Cellular Invasion and Migration.Progerin phosphorylation in interphase is lower and less mechanosensitive than lamin-A,C in iPS-derived mesenchymal stem cells.
P2860
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P2860
Hutchinson-Gilford progeria syndrome alters nuclear shape and reduces cell motility in three dimensional model substrates.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@en
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@nl
type
label
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@en
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@nl
prefLabel
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@en
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@nl
P2093
P2860
P356
P1433
P1476
Hutchinson-Gilford progeria sy ...... dimensional model substrates.
@en
P2093
Andrew D Rape
Elizabeth A Booth-Gauthier
Marion Ghibaudo
P2860
P304
P356
10.1039/C3IB20231C
P577
2013-03-01T00:00:00Z