2,3,7,8-tetrachlorodibenzo-p-dioxin impairs an insulin signaling pathway through the induction of tumor necrosis factor-alpha in adipocytes.

2,3,7,8-tetrachlorodibenzo-p-dioxin impairs an insulin signaling pathway through the induction of tumor necrosis factor-alpha in adipocytes.

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http://www.wikidata.org/entity/Q42829133
Insulin resistance and environmental pollutants: experimental evidence and future perspectivesEvaluation of the association between persistent organic pollutants (POPs) and diabetes in epidemiological studies: a national toxicology program workshop reviewAssociation of dioxin and other persistent organic pollutants (POPs) with diabetes: epidemiological evidence and new mechanisms of beta cell dysfunctionThe paradox of progress: environmental disruption of metabolism and the diabetes epidemicPersistent organic pollutants as risk factors for type 2 diabetesMetabolism Disrupting Chemicals and Metabolic DisordersEnvironmental endocrine disruption of energy metabolism and cardiovascular risk.Persistent organic pollutants and inflammatory markers in a cross-sectional study of elderly Swedish people: the PIVUS cohort.Increase in metabolic syndrome-related hospitalizations in relation to environmental sources of persistent organic pollutantsThe novel endocrine disruptor tolylfluanid impairs insulin signaling in primary rodent and human adipocytes through a reduction in insulin receptor substrate-1 levels.Inflammatory pathway genes belong to major targets of persistent organic pollutants in adipose cells.Aryl hydrocarbon receptor deficiency protects mice from diet-induced adiposity and metabolic disorders through increased energy expenditure.The hijacking of cellular signaling and the diabetes epidemic: mechanisms of environmental disruption of insulin action and glucose homeostasis.Cytokines as molecular targets for aryl hydrocarbon receptor ligands: implications for toxicity and xenobiotic detoxification.Polluted Pathways: Mechanisms of Metabolic Disruption by Endocrine Disrupting Chemicals.10-Hydroxy-2-decenoic acid, a unique medium-chain fatty acid, activates 5'-AMP-activated protein kinase in L6 myotubes and mice.Adipose Tissue as a Site of Toxin Accumulation.
P2860

2,3,7,8-tetrachlorodibenzo-p-dioxin impairs an insulin signaling pathway through the induction of tumor necrosis factor-alpha in adipocytes.

Item
http://www.wikidata.org/entity/Q42829133
description
2010 nî lūn-bûn
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2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
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2010年論文
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2010年论文
@zh-cn
name
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@en
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@nl
type
label
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@en
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@nl
prefLabel
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@en
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@nl
P1433
P1476
P2093
P304
P31
P356
P433
P478
P577
P698
P921
P356
P1433
P1476
2,3,7,8-tetrachlorodibenzo-p-d ...... is factor-alpha in adipocytes.
@en
P2093
P304
P31
P356
P433
P478
P577
P698
P921