Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.
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Comparative Multi-Epitope-Ligand-Cartography reveals essential immunological alterations in Barrett's metaplasia and esophageal adenocarcinomaTRAIL-R2: a novel apoptosis-mediating receptor for TRAIL.Generation of anti-apoptotic presenilin-2 polypeptides by alternative transcription, proteolysis, and caspase-3 cleavageFas-ligand: privilege and perilRORgamma t, a novel isoform of an orphan receptor, negatively regulates Fas ligand expression and IL-2 production in T cellsRecognition of six-transmembrane epithelial antigen of the prostate-expressing tumor cells by peptide antigen-induced cytotoxic T lymphocytesRole of calmodulin in HIV-potentiated Fas-mediated apoptosisRegulation of lymphocyte apoptosis by interferon regulatory factor 4 (IRF-4)Induction of Fas ligand expression by HIV involves the interaction of Nef with the T cell receptor zeta chainMechanisms of phosphatidylserine exposure, a phagocyte recognition signal, on apoptotic T lymphocytesLife and death of lymphocytes: a role in immunesenescenceTumor-derived exosomes in cancer progression and treatment failureDeath receptor-ligand systems in cancer, cell death, and inflammationPEDF and its roles in physiological and pathological conditions: implication in diabetic and hypoxia-induced angiogenic diseasesIL-2 sensitivity and exogenous IL-2 concentration gradient tune the productive contact duration of CD8(+) T cell-APC: a multiscale modeling studyThe relationship between apoptosis, chromatin configuration, histone modification and competence of oocytes: A study using the mouse ovary-holding stress model.Lymphocyte apoptosis during classical swine fever: implication of activation-induced cell deathThe Spliceosomal Phosphopeptide P140 Controls the Lupus Disease by Interacting with the HSC70 Protein and via a Mechanism Mediated by γδ T CellsFas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative diseaseProtein kinase C regulates FADD recruitment and death-inducing signaling complex formation in Fas/CD95-induced apoptosisSelective induction of cyclooxygenase-2 plays a role in lysophosphatidic acid regulated Fas ligand cell surface presentationHIV-1 protease processes procaspase 8 to cause mitochondrial release of cytochrome c, caspase cleavage and nuclear fragmentationIdentification of interaction partners of the cytosolic polyproline region of CD95 ligand (CD178)Characterization of Fas (Apo-1, CD95)-Fas ligand interactionTubular cell HIV-1 gp120 expression induces caspase 8 activation and apoptosisThe role of c-FLIP in modulation of CD95-induced apoptosisModeling T- and B-cell growth and differentiationPartial tolerance of subcutaneously transplanted xenogeneic tumour cell graft by Fas-mediated immunosuppressionProstaglandin E2 inhibits T cell activation-induced apoptosis and Fas-mediated cellular cytotoxicity by blockade of Fas-ligand inductionThe FAS ligand promoter polymorphism, rs763110 (-844C>T), contributes to cancer susceptibility: evidence from 19 case-control studiesc-Myc-deficient B lymphocytes are resistant to spontaneous and induced cell deathRequirement of the familial Alzheimer's disease gene PS2 for apoptosis. Opposing effect of ALG-3Bcl-2 and Fas/APO-1 regulate distinct pathways to lymphocyte apoptosisSevere destructive autoimmune lesions with aging in murine Sjögren's syndrome through Fas-mediated apoptosisT cells express a phagocyte-type NADPH oxidase that is activated after T cell receptor stimulationCaspase-dependent cleavage of the hematopoietic specific adaptor protein Gads alters signalling from the T cell receptor.Combining Fas mutation with interleukin-2 deficiency prevents Colitis and Lupus: implicating interleukin-2 for auto-reactive T cell expansion and Fas ligand for colon epithelial cell death.Fas ligand is required for the development of respiratory syncytial virus vaccine-enhanced diseaseDistinct T cell receptor signaling requirements for perforin- or FasL-mediated cytotoxicity.Lack of SHPTP1 results in src-family kinase hyperactivation and thymocyte hyperresponsiveness.
P2860
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P2860
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.
@en
type
label
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.
@en
prefLabel
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.
@en
P2093
P356
P1433
P1476
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation
@en
P2093
Ettinger R
Marshak-Rothstein A
Stanger BZ
el-Khatib M
P2888
P304
P356
10.1038/373444A0
P407
P50
P577
1995-02-01T00:00:00Z
P6179
1037666095