Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
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TRB2, a mouse Tribbles ortholog, suppresses adipocyte differentiation by inhibiting AKT and C/EBPbetaAdipogenesisModulation of gene expression by 3-iodothyronamine: genetic evidence for a lipolytic patternInterleukin-4 regulates lipid metabolism by inhibiting adipogenesis and promoting lipolysisLow intensity pulsed ultrasound (LIPUS) influences the multilineage differentiation of mesenchymal stem and progenitor cell lines through ROCK-Cot/Tpl2-MEK-ERK signaling pathwayA dual role for oncostatin M signaling in the differentiation and death of mammary epithelial cells in vivo.The role of JAK-STAT signaling in adipose tissue function.Oncostatin M maintains the hematopoietic microenvironment in the bone marrow by modulating adipogenesis and osteogenesis.Gp130 cytokines exert differential patterns of crosstalk in adipocytes both in vitro and in vivo.The gp130 receptor cytokine family: regulators of adipocyte development and function.Involvement of Visceral Adipose Tissue in Immunological Modulation of Inflammatory Cascade in PreeclampsiaNeuropoietin attenuates adipogenesis and induces insulin resistance in adipocytes.Lack of oncostatin M receptor β leads to adipose tissue inflammation and insulin resistance by switching macrophage phenotype.New insights into the roles of Stat5a/b and Stat3 in T cell development and differentiation.The enigmatic cytokine oncostatin m and roles in diseaseOncostatin m is produced in adipose tissue and is regulated in conditions of obesity and type 2 diabetes.Emerging roles of JAK-STAT signaling pathways in adipocytesIdentification of STAT target genes in adipocytes.Oncostatin m modulation of lipid storage.Cocoa tea (Camellia ptilophylla) water extract inhibits adipocyte differentiation in mouse 3T3-L1 preadipocytes.Loss of Oncostatin M Signaling in Adipocytes Induces Insulin Resistance and Adipose Tissue Inflammation in Vivo.CXCL3 positively regulates adipogenic differentiation.Retention in the Golgi apparatus and expression on the cell surface of Cfr/Esl-1/Glg-1/MG-160 are regulated by two distinct mechanisms.HRASLS3 is a PPARgamma-selective target gene that promotes adipocyte differentiationCocoa polyphenols suppress adipogenesis in vitro and obesity in vivo by targeting insulin receptor.The role of oncostatin M regulates osteoblastic differentiation of dental pulp stem cells through STAT3 pathway.Induction of osteogenesis in mesenchymal stem cells by activated monocytes/macrophages depends on oncostatin M signaling.Oncostatin M in the development of metabolic syndrome and its potential as a novel therapeutic target.Oncostatin M regulates osteogenic differentiation of murine adipose-derived mesenchymal progenitor cells through a PKCdelta-dependent mechanism.
P2860
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P2860
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@en
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@nl
type
label
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@en
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@nl
prefLabel
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@en
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@nl
P2093
P2860
P356
P1476
Oncostatin M inhibits adipogenesis through the RAS/ERK and STAT5 signaling pathways.
@en
P2093
Atsushi Miyajima
Minoru Tanaka
Takahiro Naiki
Yuichiro Miyaoka
P2860
P304
37913-37920
P356
10.1074/JBC.M606089200
P407
P577
2006-10-06T00:00:00Z