Universal absence of merlin, but not other ERM family members, in schwannomas. - Wikidata - wikimedia - TriplyDB

Universal absence of merlin, but not other ERM family members, in schwannomas.

Universal absence of merlin, but not other ERM family members, in schwannomas.

http://www.wikidata.org/entity/Q42839207

about

Cloning and characterization of SCHIP-1, a novel protein interacting specifically with spliced isoforms and naturally occurring mutant NF2 proteins Janus kinases and focal adhesion kinases play in the 4.1 band: a superfamily of band 4.1 domains important for cell structure and signal transduction The importance of nerve microenvironment for schwannoma development.Unfurling of the band 4.1, ezrin, radixin, moesin (FERM) domain of the merlin tumor suppressor p75NTR is highly expressed in vestibular schwannomas and promotes cell survival by activating nuclear transcription factor κB.mTORC1 inhibition delays growth of neurofibromatosis type 2 schwannoma.Microarray analysis of gene expression in vestibular schwannomas reveals SPP1/MET signaling pathway and androgen receptor deregulation.Molecular mechanisms promoting the pathogenesis of Schwann cell neoplasms Contribution of persistent C-Jun N-terminal kinase activity to the survival of human vestibular schwannoma cells by suppression of accumulation of mitochondrial superoxides.The ErbB inhibitors trastuzumab and erlotinib inhibit growth of vestibular schwannoma xenografts in nude mice: a preliminary study Schwann cell hyperplasia and tumors in transgenic mice expressing a naturally occurring mutant NF2 protein Global expression profile in low grade meningiomas and schwannomas shows upregulation of PDGFD, CDH1 and SLIT2 compared to their healthy tissue.The location of constitutional neurofibromatosis 2 (NF2) splice site mutations is associated with the severity of NF2.Statistical considerations for immunohistochemistry panel development after gene expression profiling of human cancers.The neurofibromatosis type 2 gene is mutated in perineurial cell tumors: a molecular genetic study of eight cases.The molecular pathogenesis of schwannomatosis, a paradigm for the co-involvement of multiple tumour suppressor genes in tumorigenesis.The neurofibromatosis type 2 gene product, merlin, reverses the F-actin cytoskeletal defects in primary human Schwannoma cells.Prospect and progress of oncolytic viruses for treating peripheral nerve sheath tumors.Schwannoma of the tongue.Neurofibromatosis type 2 tumor suppressor protein is expressed in oligodendrocytes and regulates cell proliferation and process formation.NF2/Merlin Inactivation and Potential Therapeutic Targets in Mesothelioma.

P2860

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P2860

Universal absence of merlin, but not other ERM family members, in schwannomas.

http://www.wikidata.org/entity/Q42839207

description

1997 nî lūn-bûn

@nan

1997年の論文

@ja

1997年論文

@yue

1997年論文

@zh-hant

1997年論文

@zh-hk

1997年論文

@zh-mo

1997年論文

@zh-tw

1997年论文

@wuu

1997年论文

@zh

1997年论文

@zh-cn

name

Universal absence of merlin, but not other ERM family members, in schwannomas.

@en

Universal absence of merlin, but not other ERM family members, in schwannomas.

@nl

type

label

Universal absence of merlin, but not other ERM family members, in schwannomas.

@en

Universal absence of merlin, but not other ERM family members, in schwannomas.

@nl

prefLabel

Universal absence of merlin, but not other ERM family members, in schwannomas.

@en

Universal absence of merlin, but not other ERM family members, in schwannomas.

@nl

P1433

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P1433

The American Journal of Pathology

P1476

Universal absence of merlin, but not other ERM family members, in schwannomas

@en

P2093

A O Stemmer-Rachamimov

http://www.w3.org/2001/XMLSchema#string

C Gonzalez-Agosti

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D N Louis

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D Pinney

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J A Burwick

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L B Jacoby

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L Xu

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R Beauchamp

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V Ramesh

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P2860

Ezrin has properties to self-associate at the plasma membrane

Alteration in a new gene encoding a putative membrane-organizing protein causes neuro-fibromatosis type 2

Ezrin self-association involves binding of an N-terminal domain to a normally masked C-terminal domain that includes the F-actin binding site

Regulation mechanism of ERM (ezrin/radixin/moesin) protein/plasma membrane association: possible involvement of phosphatidylinositol turnover and Rho-dependent signaling pathway

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Expression of NF2-encoded merlin and related ERM family proteins in the human central nervous system

Frequent NF2 gene transcript mutations in sporadic meningiomas and vestibular schwannomas.

High frequency of inactivating mutations in the neurofibromatosis type 2 gene (NF2) in primary malignant mesotheliomas.

Subcellular localization of moesin in dynamic filopodia, retraction fibers, and other structures involved in substrate exploration, attachment, and cell-cell contacts.

ERM (ezrin/radixin/moesin) family: from cytoskeleton to signal transduction.

P304

1649-1654

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scholarly article

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P433

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P478

151

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P50

James F. Gusella

P577

1997-12-01T00:00:00Z

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P698

9403715

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P932

1858373

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