NMDA receptor ablation on parvalbumin-positive interneurons impairs hippocampal synchrony, spatial representations, and working memory.
about
GABAergic interneuron origin of schizophrenia pathophysiology.Inhibitory interneuron deficit links altered network activity and cognitive dysfunction in Alzheimer model.GABAB-mediated rescue of altered excitatory-inhibitory balance, gamma synchrony and behavioral deficits following constitutive NMDAR-hypofunction.Energy and Potassium Ion Homeostasis during Gamma OscillationsHigh-frequency oscillations and the neurobiology of schizophreniaImpact of ketamine on neuronal network dynamics: translational modeling of schizophrenia-relevant deficits.Following the genes: a framework for animal modeling of psychiatric disordersTurning a Negative into a Positive: Ascending GABAergic Control of Cortical Activation and ArousalThe role of glutamatergic inputs onto parvalbumin-positive interneurons: relevance for schizophreniaThe role of α5 GABAA receptor agonists in the treatment of cognitive deficits in schizophreniaGamma rhythms link prefrontal interneuron dysfunction with cognitive inflexibility in Dlx5/6(+/-) mice.Inhibition of parvalbumin-expressing interneurons results in complex behavioral changes.Knockout of NMDA-receptors from parvalbumin interneurons sensitizes to schizophrenia-related deficits induced by MK-801The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophreniaParvalbumin and neuropeptide Y expressing hippocampal GABA-ergic inhibitory interneuron numbers decline in a model of Gulf War illnessDopamine D4 receptor activation increases hippocampal gamma oscillations by enhancing synchronization of fast-spiking interneuronsThe brain dynamics of linguistic computationSpatial and temporal boundaries of NMDA receptor hypofunction leading to schizophrenia.GluN2D N-Methyl-d-Aspartate Receptor Subunit Contribution to the Stimulation of Brain Activity and Gamma Oscillations by Ketamine: Implications for SchizophreniaThe NMDA receptor GluN2C subunit controls cortical excitatory-inhibitory balance, neuronal oscillations and cognitive function.Spontaneous Gamma Activity in Schizophrenia.Targeting Oxidative Stress and Aberrant Critical Period Plasticity in the Developmental Trajectory to Schizophrenia.Pyramidal cell selective ablation of N-methyl-D-aspartate receptor 1 causes increase in cellular and network excitability.Mutation of Dcdc2 in mice leads to impairments in auditory processing and memory ability.Disruption of mGluR5 in parvalbumin-positive interneurons induces core features of neurodevelopmental disorders.Parvalbumin and GAD65 interneuron inhibition in the ventral hippocampus induces distinct behavioral deficits relevant to schizophrenia.GluN2C/GluN2D subunit-selective NMDA receptor potentiator CIQ reverses MK-801-induced impairment in prepulse inhibition and working memory in Y-maze test in mice.Brain state-dependent abnormal LFP activity in the auditory cortex of a schizophrenia mouse model.How Nox2-containing NADPH oxidase affects cortical circuits in the NMDA receptor antagonist model of schizophrenia.Differential role of NR2A and NR2B subunits in N-methyl-D-aspartate receptor antagonist-induced aberrant cortical gamma oscillations.Removal of GABA(A) receptor γ2 subunits from parvalbumin neurons causes wide-ranging behavioral alterations.Ketamine alters oscillatory coupling in the hippocampus.Visual recognition memory, manifested as long-term habituation, requires synaptic plasticity in V1A negative feedback loop controls NMDA receptor function in cortical interneurons via neuregulin 2/ErbB4 signalling.The group II metabotropic glutamate receptor agonist LY354740 and the D2 receptor antagonist haloperidol reduce locomotor hyperactivity but fail to rescue spatial working memory in GluA1 knockout mice.Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells.Mice lacking the transcriptional coactivator PGC-1α exhibit alterations in inhibitory synaptic transmission in the motor cortex.Cannabinoids and Vanilloids in Schizophrenia: Neurophysiological Evidence and Directions for Basic Research.Speed controls the amplitude and timing of the hippocampal gamma rhythm.Highly energized inhibitory interneurons are a central element for information processing in cortical networks.
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P2860
NMDA receptor ablation on parvalbumin-positive interneurons impairs hippocampal synchrony, spatial representations, and working memory.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
NMDA receptor ablation on parv ...... entations, and working memory.
@en
NMDA receptor ablation on parv ...... entations, and working memory.
@nl
type
label
NMDA receptor ablation on parv ...... entations, and working memory.
@en
NMDA receptor ablation on parv ...... entations, and working memory.
@nl
prefLabel
NMDA receptor ablation on parv ...... entations, and working memory.
@en
NMDA receptor ablation on parv ...... entations, and working memory.
@nl
P50
P1433
P1476
NMDA receptor ablation on parv ...... entations, and working memory.
@en
P2093
Elke C Fuchs
Tatiana Korotkova
P304
P356
10.1016/J.NEURON.2010.09.017
P407
P577
2010-11-01T00:00:00Z